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[已撤回] 缺氧诱导的DNM1L线粒体易位增加线粒体裂变,并通过激活HK2触发肝癌细胞中的线粒体通透性转换孔开放。

[Retracted] Hypoxia‑induced mitochondrial translocation of DNM1L increases mitochondrial fission and triggers mPTP opening in HCC cells via activation of HK2.

作者信息

Cai Min, He Ping, Fang Dian-Liang

机构信息

Department of Nephrology, Yongchuan Hospital of Chongqing Medical University, Chongqing 402160, P.R. China.

Department of Gastroenterology, Yongchuan Hospital of Chongqing Medical University, Chongqing 402160, P.R. China.

出版信息

Oncol Rep. 2021 Jun;45(6). doi: 10.3892/or.2021.8059. Epub 2021 Apr 28.

Abstract

Subsequently to the publication of the above paper, the authors have retrospectively realized that they used the human normal liver cell, line L‑02, for the experiments reported in this study instead of the intended hepatocellular cell line, Huh‑7. Consequently, the results and conclusions reported in this article must be considered to lack reliability. Therefore, the authors have requested that the article be retracted from the publication. The authors apologize to the Editor and to the readership for any inconvenience caused. [the original article was published in 42: 1125‑1132, 2019; DOI: 10.3892/or.2019.7213].

摘要

在上述论文发表之后,作者们通过回顾发现,他们在本研究报告的实验中使用的是人类正常肝细胞系L-02,而非预期的肝癌细胞系Huh-7。因此,本文所报告的结果和结论必须被视为缺乏可靠性。所以,作者们已要求撤回该文章。作者们就由此造成的不便向编辑和读者表示歉意。[原文发表于《肿瘤学报道》42: 1125-1132, 2019;DOI: 10.3892/or.2019.7213]

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