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冠状窦口闭锁/异常合并永存左上腔静脉-左心室导线植入的机会和未识别的血栓栓塞性卒中来源。

The combination of coronary sinus ostial atresia/abnormalities and a small persistent left superior vena cava-Opportunity for left ventricular lead implantation and unrecognized source of thromboembolic stroke.

机构信息

Georgetown University School of Medicine, Washington, District of Columbia.

Georgetown University MedStar Heart & Vascular Institute, Washington, District of Columbia; Penn Medicine Lancaster General Health, Lancaster, Pennsylvania.

出版信息

Heart Rhythm. 2021 Jul;18(7):1064-1073. doi: 10.1016/j.hrthm.2021.05.004. Epub 2021 May 8.

Abstract

BACKGROUND

Coronary sinus (CS) ostial atresia/abnormalities prevent access to the CS from the right atrium (RA) for left ventricular (LV) lead implantation. Some patients with CS ostial abnormalities also have a small persistent left superior vena cava (sPLSVC).

OBJECTIVE

The purpose of this study was to describe CS ostial abnormalities and sPLSVC as an opportunity for LV lead implantation and unrecognized source of stroke.

METHODS

Twenty patients with CS ostial abnormalities and sPLSVC were identified. Clinical information, imaging methods, LV lead implantation techniques, and complications were summarized.

RESULTS

Forty percent had at least 1 previously unsuccessful LV lead placement. In 70%, sPLSVC was identified by catheter manipulation and contrast injection in the left brachiocephalic vein, and in 30% by levophase CS venography. In 30%, sPLSVC was associated with drainage from the CS into the left atrium (LA). When associated with CS ostial abnormalities, the sPLSVC diameter averaged 5.6 ± 3 mm. sPLSVC was used for successful LV lead implantation in 90% of cases. In 80%, the LV lead was implanted down sPLSVC, and in 20%, sPLSVC was used to access the CS from the RA. Presumably because of unrecognized drainage from the CS to the LA, 1 patient had a stroke during implantation via sPLSVC.

CONCLUSION

When CS ostial abnormalities prevent access to the CS from the RA, sPLSVC can be used to successfully implant LV leads. In some, the CS partially drains into the LA and stroke can occur spontaneously or during lead intervention. It is important to distinguish sPLSVC associated with CS ostial abnormalities from isolated PLSVC.

摘要

背景

冠状窦(CS)口狭窄/异常妨碍了从右心房(RA)进入 CS 以植入左心室(LV)导联。一些 CS 口异常的患者还伴有小的永存左上腔静脉(sPLSVC)。

目的

本研究旨在描述 CS 口异常和 sPLSVC 作为 LV 导联植入的机会和未被识别的卒中来源。

方法

共确定了 20 例 CS 口异常伴 sPLSVC 的患者。总结了临床资料、影像学方法、LV 导联植入技术和并发症。

结果

40%的患者至少有 1 次 LV 导联植入失败。在 70%的患者中,sPLSVC 通过导管操作和左头臂静脉造影剂注射发现,在 30%的患者中通过左 CS 窦期造影发现。在 30%的患者中,sPLSVC 与 CS 引流至左心房(LA)有关。当与 CS 口异常并存时,sPLSVC 直径平均为 5.6±3mm。90%的情况下,sPLSVC 被用于成功植入 LV 导联。在 80%的患者中,LV 导联经 sPLSVC 植入,在 20%的患者中,sPLSVC 被用于从 RA 进入 CS。由于未被识别的 CS 至 LA 的引流,1 例患者在通过 sPLSVC 植入时发生了卒中。

结论

当 CS 口异常妨碍了从 RA 进入 CS 时,sPLSVC 可用于成功植入 LV 导联。在某些情况下,CS 部分引流至 LA,并且卒中可自发性或在导联干预时发生。区分与 CS 口异常相关的 sPLSVC 与孤立性 PLSVC 非常重要。

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