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Developmental Alterations in Adult-Onset Neurodegenerative Disorders: Lessons from Polyglutamine Diseases.
Mov Disord. 2021 Jul;36(7):1548-1552. doi: 10.1002/mds.28657. Epub 2021 May 20.
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Targeting protein aggregation in neurodegeneration--lessons from polyglutamine disorders.
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Protective effect of chaperones on polyglutamine diseases.
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Autophagy and polyglutamine diseases.
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[Role of chromatin alterations in neurodegeneration induced by polyglutamine-expanded ataxin-7].
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[Mechanisms of neurodegeneration in polyglutamine diseases].
Rinsho Shinkeigaku. 2003 Nov;43(11):901-2.
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Current understanding on the pathogenesis of polyglutamine diseases.
Neurosci Bull. 2010 Jun;26(3):247-56. doi: 10.1007/s12264-010-0113-2.
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Therapeutic opportunities in polyglutamine disease.
Nat Med. 2001 Apr;7(4):419-23. doi: 10.1038/86486.
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[Molecular biology of polyglutamine diseases].
Postepy Hig Med Dosw. 2002;56(6):779-88.

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Bridging the gap: insights into sensorimotor deficits in NMDA receptor antibody encephalitis.
J Clin Invest. 2025 Mar 3;135(5):e188251. doi: 10.1172/JCI188251.
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Reactive Bergmann glia play a central role in spinocerebellar ataxia inflammation via the JNK pathway.
J Neuroinflammation. 2023 May 26;20(1):126. doi: 10.1186/s12974-023-02801-1.

本文引用的文献

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Glia in Neurodegeneration: The Housekeeper, the Defender and the Perpetrator.
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Huntington's Disease: Les Jeux Sont Faits?
Trends Mol Med. 2020 Oct;26(10):889-890. doi: 10.1016/j.molmed.2020.08.004. Epub 2020 Sep 4.
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Huntington's disease alters human neurodevelopment.
Science. 2020 Aug 14;369(6505):787-793. doi: 10.1126/science.aax3338. Epub 2020 Jul 16.
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The contribution of glial cells to Huntington's disease pathogenesis.
Neurobiol Dis. 2020 Sep;143:104963. doi: 10.1016/j.nbd.2020.104963. Epub 2020 Jun 25.
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Abnormal development of cerebellar-striatal circuitry in Huntington disease.
Neurology. 2020 May 5;94(18):e1908-e1915. doi: 10.1212/WNL.0000000000009364. Epub 2020 Apr 7.
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The pathobiology of perturbed mutant huntingtin protein-protein interactions in Huntington's disease.
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Abnormal brain development in child and adolescent carriers of mutant huntingtin.
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