Ugawa Y, Inoue K, Takemura T, Iwamasa T
Ann Neurol. 1986 Mar;19(3):294-7. doi: 10.1002/ana.410190313.
A 42-year-old man with adult-onset type III glycogenosis (Cori's disease) developed a gradually progressive polyneuropathy with markedly reduced activity of muscle amylo-1,6-glucosidase and glycogen accumulation within all elements of biopsied sural nerve, including axons, as shown by ultrastructural assessment.
一名42岁成年发病的III型糖原贮积病（科里氏病）男性患者出现了逐渐进展的多发性神经病，肌肉淀粉-1,6-葡萄糖苷酶活性显著降低，并且经超微结构评估显示，活检腓肠神经的所有成分（包括轴突）内均有糖原蓄积。
