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Blood insulin responses to blood glucose levels in high output sepsis and spetic shock.

作者信息

Clowes G H, Martin H, Walji S, Hirsch E, Gazitua R, Goodfellow R

出版信息

Am J Surg. 1978 Apr;135(4):577-83. doi: 10.1016/0002-9610(78)90040-5.

DOI:10.1016/0002-9610(78)90040-5
PMID:345832
Abstract
摘要

相似文献

1
Blood insulin responses to blood glucose levels in high output sepsis and spetic shock.
Am J Surg. 1978 Apr;135(4):577-83. doi: 10.1016/0002-9610(78)90040-5.
2
Carbohydrate metabolism in the baboon subjected to gram-negative (E. coli) septicemia. I. Hyperglycemia with depressed plasma insulin concentrations.狒狒在遭受革兰氏阴性(大肠杆菌)败血症时的碳水化合物代谢。I. 血糖升高伴血浆胰岛素浓度降低。
Ann Surg. 1971 Jul;174(1):91-100. doi: 10.1097/00000658-197107010-00015.
3
In vivo insulin resistance during nonlethal hypermetabolic sepsis.非致死性高代谢性脓毒症期间的体内胰岛素抵抗
Circ Shock. 1989 Jun;28(2):165-78.
4
In vivo skeletal muscle insulin resistance during E coli endotoxin shock in the dog.犬大肠杆菌内毒素休克期间的体内骨骼肌胰岛素抵抗
Circ Shock. 1981;8(4):425-33.
5
Insulin response to continuous glucose load in endotoxin shock in the dog.犬内毒素休克时对持续葡萄糖负荷的胰岛素反应。
Surg Gynecol Obstet. 1977 Aug;145(2):198-202.
6
Energy metabolism in sepsis: treatment based on different patterns in shock and high output stage.脓毒症中的能量代谢:基于休克期和高排出量期不同模式的治疗
Ann Surg. 1974 May;179(5):684-96. doi: 10.1097/00000658-197405000-00023.
7
Impaired gluconeogenesis in dogs with E. coli bacteremia.
Surgery. 1974 Oct;76(4):533-41.
8
Effects of endotoxin and E. coli shock on the metabolism of lipids and carbohydrate.内毒素和大肠杆菌休克对脂质及碳水化合物代谢的影响。
Adv Exp Med Biol. 1971 Oct;23(0):195-202. doi: 10.1007/978-1-4615-9014-9_19.
9
Glucose kinetics and pyruvate dehydrogenase activity in septic rats treated with dichloroacetate.用二氯乙酸治疗的脓毒症大鼠的葡萄糖动力学和丙酮酸脱氢酶活性
Circ Shock. 1987;23(2):131-41.
10
[Carbohydrate tolerance, insulin secretion and insulin sensitivity in acute pancreatitis].[急性胰腺炎中的碳水化合物耐受性、胰岛素分泌及胰岛素敏感性]
Z Gastroenterol. 1974 Mar;12(2):81-6.

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Anti-inflammatory effects of oestrogen mediate the sexual dimorphic response to lipid-induced insulin resistance.雌激素的抗炎作用介导了脂质诱导的胰岛素抵抗的性别二态反应。
J Physiol. 2019 Aug;597(15):3885-3903. doi: 10.1113/JP277270. Epub 2019 Jul 4.
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Stress hyperglycaemia in critically ill patients and the subsequent risk of diabetes: a systematic review and meta-analysis.危重症患者的应激性高血糖及后续糖尿病风险:一项系统评价与荟萃分析
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Relationship Between Beta Cell Dysfunction and Severity of Disease Among Critically Ill Children: A STROBE-Compliant Prospective Observational Study.危重症儿童β细胞功能障碍与疾病严重程度的关系:一项符合STROBE标准的前瞻性观察性研究。
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Saturated and unsaturated fat induce hepatic insulin resistance independently of TLR-4 signaling and ceramide synthesis in vivo.饱和脂肪和不饱和脂肪在体内独立于 TLR-4 信号和神经酰胺合成诱导肝胰岛素抵抗。
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Mechanisms for insulin resistance: common threads and missing links.胰岛素抵抗的机制:共同线索和缺失环节。
Cell. 2012 Mar 2;148(5):852-71. doi: 10.1016/j.cell.2012.02.017.