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Henna-induced Heinz bodies in glucose-6-phosphate dehydrogenase-deficient newborn.

作者信息

Khefacha Linda, Berrayana Nouha, Rouag Hatem, Benhamida Hayet, Sassi Mouna

机构信息

Biology Department of Maternity and Neonatal Medicine Center, Fattouma Bourguiba University Hospital, Monastir, Tunisia.

Neonatology Department of Maternity and Neonatal Medicine Center, Fattouma Bourguiba University Hospital, Monastir, Tunisia.

出版信息

Int J Lab Hematol. 2022 Feb;44(1):59-60. doi: 10.1111/ijlh.13713. Epub 2021 Sep 29.

DOI:10.1111/ijlh.13713
PMID:34585532
Abstract
摘要

相似文献

1
Henna-induced Heinz bodies in glucose-6-phosphate dehydrogenase-deficient newborn.葡萄糖-6-磷酸脱氢酶缺乏的新生儿中指甲花染料诱导的海因茨小体。
Int J Lab Hematol. 2022 Feb;44(1):59-60. doi: 10.1111/ijlh.13713. Epub 2021 Sep 29.
2
[Effect of ethanol on the detoxification of hydrogen peroxide induced by ascorbic acid in normal erythrocytes and those with glucose-6-phosphate dehydrogenase deficiency].[乙醇对正常红细胞及葡萄糖-6-磷酸脱氢酶缺乏红细胞中抗坏血酸诱导的过氧化氢解毒作用的影响]
Sangre (Barc). 1984;29(1):10-4.
3
Change of deformability and Heinz body formation in G6PD-deficient erythrocytes treated with 5-hydroxy-6-desmethylprimaquine.用5-羟基-6-去甲基伯氨喹治疗的葡萄糖-6-磷酸脱氢酶缺乏红细胞的变形性变化和海因茨小体形成
Blood Cells. 1989;15(2):443-52; discussion 453-4.
4
Irradiation shortens the survival time of red cells deficient in glucose-6-phosphate dehydrogenase.辐射会缩短缺乏葡萄糖-6-磷酸脱氢酶的红细胞的存活时间。
Radiat Res. 1980 Mar;81(3):473-7.
5
A novel flow cytometry-based method of analyzing Heinz bodies.
Int J Lab Hematol. 2017 Feb;39(1):68-75. doi: 10.1111/ijlh.12576. Epub 2016 Dec 15.
6
Protection by ascorbate against acetylphenylhydrazine-induced Heinz body formation in glucose-6-phosphate dehydrogenase deficient erythrocytes.抗坏血酸对葡萄糖-6-磷酸脱氢酶缺乏的红细胞中乙酰苯肼诱导的海因茨小体形成的保护作用。
Br J Haematol. 1979 Feb;41(2):245-22. doi: 10.1111/j.1365-2141.1979.tb05853.x.
7
Hemolysis in dialyzed patients: tap water-induced red blood cell metabolic deficiency.透析患者的溶血：自来水诱导的红细胞代谢缺陷。
Trans Am Soc Artif Intern Organs. 1972;18(0):301-4, 310.
8
[METHEMOGLOBINEMIA, HEMOLYSIS AND FORMATION OF HEINZ BODIES IN THE ERYTHROCYTES].
Lek Veda Zahr. 1964 Jan 17;1:1-10.
9
In vitro correction of erythrocyte glucose 6-phosphate dehydrogenase (G6PD) deficiency.红细胞葡萄糖-6-磷酸脱氢酶（G6PD）缺乏症的体外纠正
Arch Biochem Biophys. 1979 Oct 15;197(2):543-50. doi: 10.1016/0003-9861(79)90278-9.
10
CONGENITAL HEINZ-BODY ANAEMIA. FURTHER EVIDENCE ON THE CAUSE OF HEINZ-BODY PRODUCTION IN RED CELLS.先天性海因茨小体细胞贫血。关于红细胞中海因茨小体产生原因的进一步证据。
Br J Haematol. 1964 Jul;10:281-90. doi: 10.1111/j.1365-2141.1964.tb00704.x.

引用本文的文献

1
Severe Hyperbilirubinemia Secondary to Henna Application in a Neonate With G6PD Deficiency: A Case Report and Literature Review.一名患有葡萄糖-6-磷酸脱氢酶（G6PD）缺乏症的新生儿因使用指甲花而继发严重高胆红素血症：病例报告及文献综述
Cureus. 2023 Feb 2;15(2):e34540. doi: 10.7759/cureus.34540. eCollection 2023 Feb.
2
Glucose-6-Phosphate Dehydrogenase Deficiency and Neonatal Hyperbilirubinemia: Insights on Pathophysiology, Diagnosis, and Gene Variants in Disease Heterogeneity.葡萄糖-6-磷酸脱氢酶缺乏症与新生儿高胆红素血症：对疾病异质性的病理生理学、诊断及基因变异的见解
Front Pediatr. 2022 May 24;10:875877. doi: 10.3389/fped.2022.875877. eCollection 2022.