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斑块侵蚀伪装为自发性冠状动脉夹层:一例猝死。

Plaque erosion masquerading as spontaneous coronary artery dissection: A case of sudden coronary death.

机构信息

Dalio Institute of Cardiovascular Imaging, Department of Radiology, New York-Presbyterian Hospital and Weill Cornell Medicine, New York, NY, USA; Department of Radiology, Ain Shams University Hospitals, Cairo, Egypt.

Dalio Institute of Cardiovascular Imaging, Department of Radiology, New York-Presbyterian Hospital and Weill Cornell Medicine, New York, NY, USA.

出版信息

J Cardiovasc Comput Tomogr. 2022 Mar-Apr;16(2):e11-e13. doi: 10.1016/j.jcct.2021.10.001. Epub 2021 Oct 8.

DOI:10.1016/j.jcct.2021.10.001
PMID:34690087
Abstract

The three most common mechanisms of thrombosis in acute coronary syndrome (ACS) are plaque rupture, plaque erosion and calcified nodule. Plaque erosion occurs over an intact fibrous cap, commonly over pathological intimal thickening (PIT) rather than thin cap fibroatheroma (TCFA), and is more common among younger and female patients. A very rare mechanism of ACS is spontaneous coronary artery dissection (SCAD), resulting from the formation of an intramural hematoma and/or intimal disruption. We present a case of sudden coronary death with the appearance of SCAD on coronary computed tomography angiography (CCTA).

摘要

急性冠状动脉综合征(ACS)中血栓形成的三个最常见机制是斑块破裂、斑块侵蚀和钙化结节。斑块侵蚀发生在完整的纤维帽上,通常发生在病理性内膜增厚(PIT)而不是薄帽纤维粥样瘤(TCFA)上,并且在年轻和女性患者中更为常见。ACS 的一种非常罕见的机制是自发性冠状动脉夹层(SCAD),这是由于形成了壁内血肿和/或内膜破裂。我们报告了一例冠状动脉计算机断层血管造影(CCTA)显示 SCAD 的猝死病例。

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