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原发性醛固酮增多症所致冠状动脉扩张的罕见表现。

Coronary ectasia due to primary aldosteronism in an exceptional presentation.

作者信息

Rasras Hammam, Brem Falmata Laouan, Ouafi Noha El, Ismaili Nabila

机构信息

Department of Cardiology, Mohammed VI University Hospital of Oujda, Mohammed First University of Oujda, Morocco.

Laboratory of Epidemiology, Clinical Research and Public Health, Faculty of Medicine and Pharmacy, Mohammed the First University of Oujda, Morocco.

出版信息

Glob Cardiol Sci Pract. 2021 Oct 30;2021(3):e202122. doi: 10.21542/gcsp.2021.22.

DOI:10.21542/gcsp.2021.22
PMID:34805380
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8587354/
Abstract

Primary aldosteronism as a cause of coronary ectasia has been reported only once in the literature and was associated with an aortic aneurysm. Here, we report a second presentation in our cardiology department - a 59-year-old female patient who was admitted for unstable angina. Coronary angiography revealed an ectasia of two major coronary arteries. An etiological assessment revealed an idiopathic primary aldosteronism.

摘要

原发性醛固酮增多症作为冠状动脉扩张的病因在文献中仅被报道过一次，且与主动脉瘤相关。在此，我们报告我院心内科的第二例病例——一名59岁女性患者因不稳定型心绞痛入院。冠状动脉造影显示两条主要冠状动脉扩张。病因评估显示为特发性原发性醛固酮增多症。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/df4d/8587354/86b52c45eaae/gcsp-2021-3-e202122-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/df4d/8587354/cce93b396234/gcsp-2021-3-e202122-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/df4d/8587354/86b52c45eaae/gcsp-2021-3-e202122-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/df4d/8587354/cce93b396234/gcsp-2021-3-e202122-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/df4d/8587354/86b52c45eaae/gcsp-2021-3-e202122-g002.jpg

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本文引用的文献

1
Aneurysmal coronary artery disease: An overview.动脉瘤性冠状动脉疾病：概述
Glob Cardiol Sci Pract. 2017 Oct 31;2017(3):e201726. doi: 10.21542/gcsp.2017.26.
2
Coronary Artery Ectasia-A Review of Current Literature.冠状动脉扩张——当前文献综述
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Coronary artery aneurysms: a review of the natural history, pathophysiology, and management.冠状动脉瘤：自然病史、病理生理学及治疗综述
Cardiol Rev. 2008 Nov-Dec;16(6):301-4. doi: 10.1097/CRD.0b013e3181852659.
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