β-内酰胺酶去阻遏阴沟肠杆菌中头孢噻肟和头孢曲松耐药性的水解模型 - Suppr | 超能文献

Hydrolytic model for cefotaxime and ceftriaxone resistance in beta-lactamase-derepressed Enterobacter cloacae.

作者信息

Livermore D M, Riddle S J, Davy K W

出版信息

J Infect Dis. 1986 Mar;153(3):619-22. doi: 10.1093/infdis/153.3.619.

DOI:10.1093/infdis/153.3.619

Abstract

摘要

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Hydrolytic model for cefotaxime and ceftriaxone resistance in beta-lactamase-derepressed Enterobacter cloacae.β-内酰胺酶去阻遏阴沟肠杆菌中头孢噻肟和头孢曲松耐药性的水解模型

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引用本文的文献

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Emergence of resistance during beta-lactam therapy of gram-negative infections. Bacterial mechanisms and medical responses.革兰氏阴性菌感染β-内酰胺治疗期间耐药性的出现。细菌机制与医学应对措施。

Drugs. 1988;35 Suppl 2:22-8. doi: 10.2165/00003495-198800352-00007.

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Resistance to third generation cephalosporins: the current situation.对第三代头孢菌素的耐药性：现状

Infection. 1989 Sep-Oct;17(5):333-7. doi: 10.1007/BF01650724.

3

Imipenem- and meropenem-resistant mutants of Enterobacter cloacae and Proteus rettgeri lack porins.阴沟肠杆菌和雷氏变形杆菌对亚胺培南和美罗培南耐药的突变体缺乏孔蛋白。

Antimicrob Agents Chemother. 1991 Jun;35(6):1174-80. doi: 10.1128/AAC.35.6.1174.