Smith P F, Darlington C L, Curthoys I S
Neurosci Lett. 1986 Apr 11;65(2):209-13. doi: 10.1016/0304-3940(86)90306-x.
The rapid recovery from the postural and ocular motor asymmetries produced by unilateral vestibular damage (vestibular compensation) has been presented as an example of plasticity in the central nervous system. A recent model (J. Neurophysiol., 51 (1984) 242-259) has identified the fibers joining the two vestibular nuclei in the brainstem (the vestibular commissures) as the site of the plastic changes. We report that in guinea pigs, compensation of postural symptoms still occurs after sectioning these commissural fibers. We suggest that a number of mechanisms may be responsible for vestibular compensation, including some which are independent of the vestibular commissures.
单侧前庭损伤所导致的姿势和眼球运动不对称的快速恢复（前庭代偿），已被视为中枢神经系统可塑性的一个例子。最近的一个模型（《神经生理学杂志》，51卷（1984年）242 - 259页）已确定，脑干中连接两个前庭核的纤维（前庭连合）是发生可塑性变化的部位。我们报告称，在豚鼠中，切断这些连合纤维后，姿势症状仍会出现代偿。我们认为，前庭代偿可能由多种机制引起，包括一些独立于前庭连合的机制。
