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Letter to the Editor regarding the article 'ER stress-related protein, CHOP, may serve as a biomarker of mechanical asphyxia: a primary study' by Hu and colleagues.

作者信息

Chighine Alberto, Locci Emanuela, Ferino Giulio, De-Giorgio Fabio, d'Aloja Ernesto

机构信息

Department of Medical Sciences and Public Health, Section of Legal Medicine, University of Cagliari, Cagliari, Italy.

Department of Health Surveillance and Bioethics, Section of Legal Medicine, Catholic University of Rome, Rome, Italy.

出版信息

Int J Legal Med. 2022 May;136(3):969-970. doi: 10.1007/s00414-022-02808-y. Epub 2022 Mar 1.

DOI:10.1007/s00414-022-02808-y
PMID:35233642
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9005414/
Abstract
摘要

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Letter to the Editor regarding the article 'ER stress-related protein, CHOP, may serve as a biomarker of mechanical asphyxia: a primary study' by Hu and colleagues.致编辑的信：关于胡及其同事所著文章《内质网应激相关蛋白CHOP可能作为机械性窒息的生物标志物：一项初步研究》
Int J Legal Med. 2022 May;136(3):969-970. doi: 10.1007/s00414-022-02808-y. Epub 2022 Mar 1.
2
ER stress-related protein, CHOP, may serve as a biomarker of mechanical asphyxia: a primary study.内质网应激相关蛋白 CHOP 可作为机械性窒息的生物标志物：初步研究
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3
[Effects of erythropoietin on cardiomyocyte apoptosis and endoplasmic reticulum stress-related proteins in neonatal rats with asphyxia].
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DUSP1 and KCNJ2 mRNA upregulation can serve as a biomarker of mechanical asphyxia-induced death in cardiac tissue.双特异性磷酸酶1（DUSP1）和内向整流型钾通道2（KCNJ2）信使核糖核酸（mRNA）的上调可作为心脏组织中机械性窒息所致死亡的生物标志物。
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Regulation of the cerebrovascular smooth muscle cell phenotype by mitochondrial oxidative injury and endoplasmic reticulum stress in simulated microgravity rats via the PERK-eIF2α-ATF4-CHOP pathway.模拟微重力大鼠通过 PERK-eIF2α-ATF4-CHOP 通路介导的线粒体氧化损伤和内质网应激对脑血管平滑肌细胞表型的调控。
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Inhibition of phosphatidylcholine synthesis induces expression of the endoplasmic reticulum stress and apoptosis-related protein CCAAT/enhancer-binding protein-homologous protein (CHOP/GADD153).抑制磷脂酰胆碱合成可诱导内质网应激及凋亡相关蛋白CCAAT/增强子结合蛋白同源蛋白（CHOP/GADD153）的表达。
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ER stress activating ATF4/CHOP-TNF-α signaling pathway contributes to alcohol-induced disruption of osteogenic lineage of multipotential mesenchymal stem cell.内质网应激激活ATF4/CHOP-TNF-α信号通路，促使酒精诱导的多能间充质干细胞成骨谱系破坏。
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Identification of the miRNA-3185/CYP4A11 axis in cardiac tissue as a biomarker for mechanical asphyxia.鉴定心脏组织中的 miRNA-3185/CYP4A11 轴作为机械性窒息的生物标志物。
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G6PC3, ALDOA and CS induction accompanies mir-122 down-regulation in the mechanical asphyxia and can serve as hypoxia biomarkers.在机械性窒息中，G6PC3、ALDOA和CS的诱导伴随着mir-122的下调，并且可作为缺氧生物标志物。
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本文引用的文献

1
Correction to: A stepwise strategy to distinguish menstrual blood from peripheral blood by Fisher's discriminant function.
Int J Legal Med. 2022 May;136(3):971-972. doi: 10.1007/s00414-022-02805-1.
2
Metabolomics improves the histopathological diagnosis of asphyxial deaths: an animal proof-of-concept model.代谢组学提高窒息性死亡的组织病理学诊断：动物概念验证模型。
Sci Rep. 2021 May 12;11(1):10102. doi: 10.1038/s41598-021-89570-0.
3
Role of Mitochondrial Calcium and the Permeability Transition Pore in Regulating Cell Death.线粒体钙和通透性转换孔在调节细胞死亡中的作用。
Circ Res. 2020 Jan 17;126(2):280-293. doi: 10.1161/CIRCRESAHA.119.316306. Epub 2020 Jan 16.
4
Krebs Cycle Reimagined: The Emerging Roles of Succinate and Itaconate as Signal Transducers.克雷布斯循环再思考：琥珀酸和衣康酸作为信号转导物的新兴作用。
Cell. 2018 Aug 9;174(4):780-784. doi: 10.1016/j.cell.2018.07.030.
5
Metabolomics profiling reveals different patterns in an animal model of asphyxial and dysrhythmic cardiac arrest.代谢组学分析揭示了窒息性和心律失常性心脏骤停动物模型中的不同模式。
Sci Rep. 2017 Nov 29;7(1):16575. doi: 10.1038/s41598-017-16857-6.
6
Moving Forwards by Blocking Back-Flow: The Yin and Yang of MI Therapy.通过阻止逆流向前迈进：心肌梗死治疗的阴阳之道
Circ Res. 2016 Mar 4;118(5):898-906. doi: 10.1161/CIRCRESAHA.115.306569.