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孤儿核受体 COUP-TFII 的异构体差异调节胰腺癌的进展。

Isoforms of the orphan nuclear receptor COUP‑TFII differentially modulate pancreatic cancer progression.

机构信息

Gastroenterology Research Unit, Department of Experimental and Clinical Biomedical Sciences 'Mario Serio', University of Florence, I-50134 Florence, Italy.

Core Research Laboratory, Institute for Cancer Research and Prevention, I-50139 Florence, Italy.

出版信息

Int J Oncol. 2022 May;60(5). doi: 10.3892/ijo.2022.5345. Epub 2022 Mar 29.

DOI:10.3892/ijo.2022.5345
PMID:35348189
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8997336/
Abstract

The expression of the nuclear receptor transcription factor (TF) COUP‑TFII is broadly associated with cell differentiation and cancer development, including of pancreatic ductal adenocarcinoma (PDAC), a devastating disease with one of the poorest prognoses among cancers worldwide. Recent studies have started to investigate the pathological and physiological roles of a novel COUP‑TFII isoform (COUP‑TFII_V2) that lacks the DNA‑binding domain. As the role of the canonical COUP‑TFII in PDAC was previously demonstrated, the present study evaluated whether COUP‑TFII_V2 may have a functional role in PDAC. It was demonstrated that COUP‑TFII_V2 naturally occurs in PDAC cells and in primary samples, where its expression is consistent with shorter overall survival and peripheral invasion. Of note, COUP‑TFII_V2, exhibiting nuclear and cytosolic expression, is linked to epithelial to mesenchymal transition (EMT) and cancer progression, as confirmed by nude mouse experiments. The present results demonstrated that COUP‑TFII_V2 distinctively regulates the EMT of PDAC and, similarly to its sibling, it is associated with tumor aggressiveness. The two isoforms have both overlapping and exclusive functions that cooperate with cancer growth and dissemination. By studying how PDAC cells switch from one isoform to the other, novel insight into cancer biology was gained, indicating that this receptor may serve as a novel possible target for PDAC management.

摘要

核受体转录因子(TF)COUP-TFII 的表达广泛与细胞分化和癌症发展相关,包括胰腺导管腺癌(PDAC),这是一种毁灭性疾病,其预后在全球癌症中最差之一。最近的研究开始探讨一种新型 COUP-TFII 同工型(COUP-TFII_V2)的病理和生理作用,该同工型缺乏 DNA 结合域。由于先前已经证明了典型的 COUP-TFII 在 PDAC 中的作用,因此本研究评估了 COUP-TFII_V2 是否可能在 PDAC 中具有功能作用。研究表明,COUP-TFII_V2 自然存在于 PDAC 细胞和原发性样本中,其表达与总生存期和周围浸润较短相一致。值得注意的是,COUP-TFII_V2 表现出核和细胞质表达,与上皮间质转化(EMT)和癌症进展相关,这通过裸鼠实验得到了证实。本研究结果表明,COUP-TFII_V2 可明显调节 PDAC 的 EMT,并且与它的同源物一样,与肿瘤侵袭性相关。这两种同工型具有重叠和独特的功能,与癌症的生长和扩散合作。通过研究 PDAC 细胞如何从一种同工型切换到另一种同工型,对癌症生物学有了新的认识,表明该受体可能成为 PDAC 治疗的新的可能靶标。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/673cb2190929/IJO-60-05-05345-g06.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/fb738e9b8656/IJO-60-05-05345-g00.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/a6638a377391/IJO-60-05-05345-g01.jpg
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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/fda9561b1f9c/IJO-60-05-05345-g04.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/e71597086f13/IJO-60-05-05345-g05.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/673cb2190929/IJO-60-05-05345-g06.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/fb738e9b8656/IJO-60-05-05345-g00.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/a6638a377391/IJO-60-05-05345-g01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/7c756080f979/IJO-60-05-05345-g02.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/f4227d62ca96/IJO-60-05-05345-g03.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/fda9561b1f9c/IJO-60-05-05345-g04.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/e71597086f13/IJO-60-05-05345-g05.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/cb65/8997336/673cb2190929/IJO-60-05-05345-g06.jpg

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