Malinova I V, Miasnik M N
Radiobiologiia. 1986 Sep-Oct;26(5):611-5.
A relative contribution of photoreactivated (modified by visible light) and non-photoreactivated (modified by temperature) damages to UV-irradiated (250-334 nm) E. coli B cells was estimated. The contribution of damages modified by temperature to a lethal effect of UV-radiation was invariable within the range from 250 to 334 nm. The photoreactivation of E. coli B cells was also independent of lambda-inactivating UV-light within 250-313 nm, and its value exceeded that of the wild-type E. coli WP2 which did not vary by the mode of UV-damages repair. Moreover, in contrast to E. coli B. cells, the value of the photoreactivation of E. coli WP2 decreased, as lambda-inactivating UV-light increased from 250 to 313 nm.
估算了光活化（由可见光修饰）和非光活化（由温度修饰）损伤对紫外线照射（250 - 334纳米）的大肠杆菌B细胞的相对贡献。在250至334纳米范围内，由温度修饰的损伤对紫外线辐射致死效应的贡献是不变的。大肠杆菌B细胞的光复活作用在250 - 313纳米范围内也与λ灭活紫外线无关，并且其值超过了野生型大肠杆菌WP2，后者不会因紫外线损伤修复模式而变化。此外，与大肠杆菌B细胞相反，随着λ灭活紫外线从250纳米增加到313纳米，大肠杆菌WP2的光复活值下降。
