• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

Definite diagnosis of plasma prekallikrein deficiency should not be based exclusively on shortening of the aPTT upon prolonged pre-incubation.

作者信息

Adenaeuer Anke, Barco Stefano, Trinchero Alice, Lackner Karl J, Lämmle Bernhard, Rossmann Heidi

机构信息

Center for Thrombosis and Hemostasis, University Medical Center Mainz, Johannes Gutenberg University, Mainz, Germany.

Institute for Clinical Chemistry and Laboratory Medicine, University Medical Center Mainz, Johannes Gutenberg University, Mainz, Germany.

出版信息

Int J Lab Hematol. 2022 Aug;44(4):e179-e180. doi: 10.1111/ijlh.13841. Epub 2022 Apr 4.

DOI:10.1111/ijlh.13841
PMID:35377536
Abstract
摘要

相似文献

1
Definite diagnosis of plasma prekallikrein deficiency should not be based exclusively on shortening of the aPTT upon prolonged pre-incubation.血浆前激肽释放酶缺乏症的明确诊断不应仅基于长时间预孵育后活化部分凝血活酶时间(aPTT)的缩短。
Int J Lab Hematol. 2022 Aug;44(4):e179-e180. doi: 10.1111/ijlh.13841. Epub 2022 Apr 4.
2
Prekallikrein deficiency: the characteristic normalization of the severely prolonged aPTT following increased preincubation time is due to autoactivation of factor XII.前激肽释放酶缺乏症:预孵育时间延长后,严重延长的活化部分凝血活酶时间(aPTT)出现特征性正常化是由于因子 XII 的自动激活。
Thromb Res. 2002 Mar 15;105(6):463-70. doi: 10.1016/s0049-3848(02)00045-2.
3
A Rare Cause of Isolated Prolonged Activated Partial Thromboplastin Time: An Overview of Prekallikrein Deficiency and the Contact System.罕见的孤立性延长部分凝血活酶时间原因:激肽释放酶原缺乏和接触系统概述。
J Investig Med High Impact Case Rep. 2021 Jan-Dec;9:23247096211012187. doi: 10.1177/23247096211012187.
4
Prolonged activated partial thromboplastin time due to plasma prekallikrein deficiency: a case study and literature review on its clinical significance.血浆前激肽释放酶缺乏导致活化部分凝血活酶时间延长:一项病例研究及其临床意义的文献综述
Blood Coagul Fibrinolysis. 2019 Sep;30(6):300-303. doi: 10.1097/MBC.0000000000000837.
5
Prolonged activated partial thromboplastin time and deficiency of high molecular weight kininogen in brown Norway rat mutant (Katholiek strain).棕色挪威大鼠突变体(天主教菌株)的活化部分凝血活酶时间延长和高分子量激肽原缺乏。
Thromb Res. 1984 Feb 15;33(4):371-7. doi: 10.1016/0049-3848(84)90076-8.
6
Prekallikrein deficiency.前激肽释放酶缺乏症
J Pediatr Oncol Nurs. 2013 Jul-Aug;30(4):198-204. doi: 10.1177/1043454213487436. Epub 2013 Apr 29.
7
Prekallikrein deficiency in a 15-year-old boy with Ménière's disease: a case report.一名患梅尼埃病的15岁男孩的前激肽释放酶缺乏症:病例报告
Acta Clin Belg. 2017 Aug;72(4):274-277. doi: 10.1080/17843286.2016.1227907. Epub 2016 Sep 10.
8
Long-term safety outcomes of prekallikrein (Fletcher factor) deficiency: A systematic literature review of case reports.遗传性高分子量激肽原缺乏症(Fletcher 因子缺乏症)的长期安全性结局:病例报告的系统文献回顾。
Allergy Asthma Proc. 2020 Jan 17;41(1):10-18. doi: 10.2500/aap.2020.41.190005.
9
Prekallikrein deficiency in a kindred with kininogen deficiency and Fitzgerald trait clotting defect. Evidence that high molecular weight kininogen and prekallikrein exist as a complex in normal human plasma.一个家族中激肽释放酶原缺乏伴激肽原缺乏和菲茨杰拉德特征性凝血缺陷。证据表明高分子量激肽原和激肽释放酶原在正常人血浆中以复合物形式存在。
J Clin Invest. 1977 Sep;60(3):571-83. doi: 10.1172/JCI108809.
10
Prekallikrein deficiency in a family of miniature horses.迷你马家族中的前激肽释放酶缺乏症。
Am J Vet Res. 1986 Nov;47(11):2464-7.

引用本文的文献

1
Targeting the Contact Pathway of Coagulation for the Prevention and Management of Medical Device-Associated Thrombosis.针对凝血接触途径的医疗设备相关血栓预防和管理。
Semin Thromb Hemost. 2024 Oct;50(7):989-997. doi: 10.1055/s-0043-57011. Epub 2023 Apr 12.