Harada Ayumi, Morisaki Koichi, Kurose Shun, Yoshino Shinichiro, Yamashita Sho, Furuyama Tadashi, Mori Masaki
Department of Surgery and Science, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Fukuoka, Japan.
Ann Vasc Dis. 2022 Mar 25;15(1):45-48. doi: 10.3400/avd.cr.21-00019.
We report a case of an 83-year-old man with a ruptured internal iliac artery (IIA) aneurysm after endovascular repair, which was treated via the ligation of IIA and tight suture of the aneurysm sac. Although there were no findings of obvious endoleak after endovascular treatment, the IIA aneurysm increased in size and eventually ruptured. We presumed that pressure to IIA aneurysm via the embolized IIA led to rupture. Aneurysm sac expansion may lead to a rupture despite no endoleak being detected; therefore, close follow-up or re-intervention must be considered. Tight embolization of IIA may prevent endotension in the same case.
我们报告一例83岁男性患者，在血管腔内修复术后发生髂内动脉（IIA）动脉瘤破裂，通过结扎IIA和紧密缝合动脉瘤囊进行治疗。尽管血管腔内治疗后未发现明显内漏，但IIA动脉瘤仍增大并最终破裂。我们推测，经栓塞的IIA对IIA动脉瘤的压力导致了破裂。尽管未检测到内漏，但动脉瘤囊扩张仍可能导致破裂；因此，必须考虑密切随访或再次干预。在同一病例中，对IIA进行紧密栓塞可能预防内张力。
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