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A tale of 2 torsades: How to approach a patient with torsades de pointes and distinguish between classical and pseudo-torsades de pointes.

作者信息

Scheinman Melvin, Bunch T Jared, Singh Mohita

机构信息

Division of Cardiology, Section on Electrophysiology, University of California San Francisco, San Francisco, California.

Division of Cardiology, Department of Internal Medicine, University of Utah, Utah.

出版信息

HeartRhythm Case Rep. 2022 Apr 15;8(4):305-308. doi: 10.1016/j.hrcr.2022.03.007. eCollection 2022 Apr.

DOI:10.1016/j.hrcr.2022.03.007
PMID:35497484
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9039100/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6651/9039100/0fb01e475211/gr5.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6651/9039100/d698a1a7da2b/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6651/9039100/b20197e774e3/gr2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6651/9039100/789258a77c72/gr3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6651/9039100/1d58951dc357/gr4.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6651/9039100/0fb01e475211/gr5.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6651/9039100/d698a1a7da2b/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6651/9039100/b20197e774e3/gr2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6651/9039100/789258a77c72/gr3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6651/9039100/1d58951dc357/gr4.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6651/9039100/0fb01e475211/gr5.jpg

相似文献

1
A tale of 2 torsades: How to approach a patient with torsades de pointes and distinguish between classical and pseudo-torsades de pointes.两例尖端扭转型室速的故事:如何诊治尖端扭转型室速患者并区分经典型与假性尖端扭转型室速。
HeartRhythm Case Rep. 2022 Apr 15;8(4):305-308. doi: 10.1016/j.hrcr.2022.03.007. eCollection 2022 Apr.
2
A canine model of torsades de pointes.
Pacing Clin Electrophysiol. 1988 Dec;11(12):2235-45. doi: 10.1111/j.1540-8159.1988.tb05990.x.
3
Monophasic action potentials in patients with torsades de pointes.
Jpn Circ J. 1994 Apr;58(4):248-58. doi: 10.1253/jcj.58.248.
4
QT prolongation and Torsades de Pointes in patients previously treated with anthracyclines.曾接受蒽环类药物治疗的患者出现QT间期延长和尖端扭转型室速。
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Efficacy and proarrhythmia with the use of d,l-sotalol for sustained ventricular tachyarrhythmias.使用消旋索他洛尔治疗持续性室性快速心律失常的疗效及促心律失常作用。
J Cardiovasc Pharmacol. 1997 Mar;29(3):373-81. doi: 10.1097/00005344-199703000-00011.
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Drug-related torsades de pointes in the isolated rabbit heart: comparison of clofilium, d,l-sotalol, and erythromycin.离体兔心脏中与药物相关的尖端扭转型室速:氯非铵、消旋索他洛尔和红霉素的比较。
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Torsades de pointes: arrhythmia, syndrome, or chimera? A perspective in the light of the Lambeth Conventions.尖端扭转型室性心动过速:心律失常、综合征还是异源嵌合体?基于兰贝斯会议的观点
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Baseline values and sotalol-induced changes of ventricular repolarization duration, heterogeneity, and instability in patients with a history of drug-induced torsades de pointes.有药物性尖端扭转型室性心动过速病史患者的基线值以及索他洛尔引起的心室复极持续时间、异质性和不稳定性的变化。
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K201, a multi-channel blocker, inhibits clofilium-induced torsades de pointes and attenuates an increase in repolarization.K201是一种多通道阻滞剂,可抑制氯非铵诱发的尖端扭转型室速,并减弱复极化的增加。
Eur J Pharmacol. 2007 Jan 19;555(1):54-60. doi: 10.1016/j.ejphar.2006.10.005. Epub 2006 Oct 17.
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The effect of intravenous haloperidol on QT interval dispersion in critically ill patients: comparison with QT interval prolongation for assessment of risk of Torsades de Pointes.静脉注射氟哌啶醇对重症患者QT间期离散度的影响:与QT间期延长比较以评估尖端扭转型室速风险
J Clin Pharmacol. 2001 Dec;41(12):1310-8. doi: 10.1177/00912700122012896.

本文引用的文献

1
Long QT Syndrome and Torsade de Pointes Ultimately Treated With Quinidine: Introducing the Concept of Pseudo-Torsade de Pointes.最终用奎尼丁治疗的长QT综合征和尖端扭转型室速:引入假性尖端扭转型室速的概念。
Circulation. 2021 Jul 6;144(1):85-89. doi: 10.1161/CIRCULATIONAHA.121.054991.
2
Polymorphic ventricular tachycardia, ischaemic ventricular fibrillation, and torsade de pointes: importance of the QT and the coupling interval in the differential diagnosis.多形性室性心动过速、缺血性心室颤动和尖端扭转型室性心动过速:QT 和偶联间期在鉴别诊断中的重要性。
Eur Heart J. 2021 Oct 7;42(38):3965-3975. doi: 10.1093/eurheartj/ehab138.
3
Cardiac ryanodine receptor calcium release deficiency syndrome.
心脏兰尼碱受体钙释放缺陷综合征。
Sci Transl Med. 2021 Feb 3;13(579). doi: 10.1126/scitranslmed.aba7287.
4
Role of the Purkinje system in heritable arrhythmias.浦肯野系统在遗传性心律失常中的作用。
Heart Rhythm. 2019 Jul;16(7):1121-1126. doi: 10.1016/j.hrthm.2019.01.034. Epub 2019 Feb 2.
5
Pleomorphic ventricular tachycardia and risk for sudden cardiac death.
Circ Arrhythm Electrophysiol. 2011 Feb;4(1):2-4. doi: 10.1161/CIRCEP.110.960443.
6
Mapping and ablation of idiopathic ventricular fibrillation.特发性室性心动过速的标测与消融
Circulation. 2002 Aug 20;106(8):962-7. doi: 10.1161/01.cir.0000027564.55739.b1.
7
Effects of electrophysiologic-guided therapy with Class IA antiarrhythmic drugs on the long-term outcome of patients with idiopathic ventricular fibrillation with or without the Brugada syndrome.ⅠA类抗心律失常药物电生理引导治疗对伴或不伴Brugada综合征的特发性室颤患者长期预后的影响
J Cardiovasc Electrophysiol. 1999 Oct;10(10):1301-12. doi: 10.1111/j.1540-8167.1999.tb00183.x.
8
Short-coupled variant of torsade de pointes. A new electrocardiographic entity in the spectrum of idiopathic ventricular tachyarrhythmias.尖端扭转型室速的短联律变异型。特发性室性快速心律失常谱系中的一种新的心电图实体。
Circulation. 1994 Jan;89(1):206-15. doi: 10.1161/01.cir.89.1.206.
9
[Ventricular tachycardia with 2 variable opposing foci].[伴有两个可变的相反起源点的室性心动过速]
Arch Mal Coeur Vaiss. 1966 Feb;59(2):263-72.