Cruz Martínez A
Eur Neurol. 1987;26(3):189-92. doi: 10.1159/000116332.
A 14-year-old girl developed right peroneal nerve palsy following diet for weight reduction. Peroneal neuropathy started after sitting and leg-crossing. The main electrophysiologic findings showed a severe slowing of conduction velocity in the above-to-below capitulum fibulae segment and a striking reduction in the size of the compound motor action potential when the nerve was stimulated above the knee. These features are consistent with focal demyelination and conduction block of the nerve fibres. The left peroneal nerve showed the same results but less severe. There were no electrophysiological signs of subclinical polyneuropathy. Examination after 3 months was normal. The clinical and electrophysiological features in this case support the compressive origin of the peroneal neuropathy in slimmer's paralysis.
一名14岁女孩在进行减肥饮食后出现右腓总神经麻痹。腓总神经病变始于久坐和交叉双腿后。主要电生理检查结果显示,在腓骨小头上下段,神经传导速度严重减慢,当在膝盖上方刺激神经时,复合运动动作电位的大小显著降低。这些特征与神经纤维的局灶性脱髓鞘和传导阻滞相符。左侧腓总神经也有同样的结果,但程度较轻。没有亚临床多发性神经病的电生理迹象。3个月后的检查结果正常。该病例的临床和电生理特征支持了减肥者麻痹中腓总神经病变的压迫性起源。