Retraction Note: Mitochondrial aldehyde dehydrogenase (ALDH2) protects against streptozotocin-induced diabetic cardiomyopathy: role of GSK3β and mitochondrial function.
作者信息
Zhang Yingmei, Babcock Sara A, Hu Nan, Maris Jacalyn R, Wang Haichang, Ren Jun
机构信息
Department of Cardiology, Xijing Hospital, Fourth Military Medical University, Xi'an, 710032, China.
Center for Cardiovascular Research and Alternative Medicine, University of Wyoming College of Health Sciences, Laramie, WY, 82071, USA.
出版信息
BMC Med. 2022 Jul 8;20(1):248. doi: 10.1186/s12916-022-02455-5.
Abstract
摘要
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Retraction Note: Mitochondrial aldehyde dehydrogenase (ALDH2) protects against streptozotocin-induced diabetic cardiomyopathy: role of GSK3β and mitochondrial function.撤稿说明：线粒体醛脱氢酶（ALDH2）对链脲佐菌素诱导的糖尿病性心肌病具有保护作用：糖原合成酶激酶3β（GSK3β）和线粒体功能的作用
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本文引用的文献
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RETRACTED: Thapsigargin triggers cardiac contractile dysfunction via NADPH oxidase-mediated mitochondrial dysfunction: Role of Akt dephosphorylation.撤回：他帕昔琼通过 NADPH 氧化酶介导的线粒体功能障碍引发心脏收缩功能障碍：Akt 去磷酸化的作用。
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Activation of Akt rescues endoplasmic reticulum stress-impaired murine cardiac contractile function via glycogen synthase kinase-3β-mediated suppression of mitochondrial permeation pore opening.通过糖原合酶激酶-3β介导的抑制线粒体通透转换孔开放，Akt 的激活挽救内质网应激损伤的小鼠心肌收缩功能。
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Advanced glycation endproduct (AGE) accumulation and AGE receptor (RAGE) up-regulation contribute to the onset of diabetic cardiomyopathy.晚期糖基化终产物（AGE）的积累和 AGE 受体（RAGE）的上调导致糖尿病心肌病的发生。
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