Niewiadomska M, Piłkowska E, Wochnik-Dyjas D
Neurol Neurochir Pol. 1986 Sep-Oct;20(5):453-60.
A group of 39 patients with alcoholic polyneuropathy (Alc) and another group of 33 patients with carbon disulphide polyneuropathy were studied by electrophysiological methods using an identical programme, and the results were compared. In both groups evidence was found of damage to the peripheral neuron of the type of axonal neuropathy with signs of axonal loss, with moderate slowing down of conduction velocity and with associated signs of regeneration. The dynamics of changes in the Alc group was greater. The cause of a slower rate of changes in the CS2 group seems to be related to a more gradual exposure to the damaging agent, while in alcohol-dependence the toxic agent has a more acute action, with remissions during abstinence and exacerbations during increased alcohol consumption.
采用相同方案，通过电生理方法对一组39例酒精性多发性神经病（Alc）患者和另一组33例二硫化碳性多发性神经病患者进行了研究，并对结果进行了比较。在两组中均发现有轴索性神经病类型的周围神经元损伤证据，伴有轴突丧失迹象、传导速度中度减慢以及相关的再生迹象。Alc组变化的动态更为明显。CS2组变化速率较慢的原因似乎与更逐渐接触损伤因子有关，而在酒精依赖中，毒性因子作用更为急性，戒酒时缓解，饮酒增加时加重。
