Nishimoto Takashi, Nishii Nobuhiro, Asada Saori, Nakagawa Koji, Morita Hiroshi, Ito Hiroshi
Department of Cardiovascular Medicine, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan.
Department of Cardiovascular Therapeutics, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan.
J Cardiol Cases. 2021 Oct 23;25(4):218-224. doi: 10.1016/j.jccase.2021.09.012. eCollection 2022 Apr.
Catheter ablation (CA) of ventricular tachycardia (VT) after repair of congenital heart disease may be difficult because of complex anatomy and sometimes unmappable VT. Here, we report a 41-year-old woman with successful CA of unmappable VT in a patient with complete transposition of the great arteries after Rastelli repair. Clinical VT was induced by programmed electrical stimulation, when the mapping catheter was placed at the high anterior right ventricular outflow tract (RVOT). During VT, the local potential at the high anterior RVOT under the right ventricle (RV) - pulmonary artery (PA) conduit was equal to that at the timing of onset of QRS. The VT was unmappable because the hemodynamics deteriorated. Pace mapping was also tried at the aortic cusp and the left ventricular outflow tract (LVOT). Fractionated potential during sinus rhythm was observed at the noncoronary cusp, and the paced QRS morphology at this site was similar to that of the clinical VT, with a delay of 55 ms from pacing to the onset of QRS. However, mapping at the LVOT was impossible due to the difficulty of catheter manipulation. Radiofrequency energy was successfully applied at the noncoronary cusp and the high anterior RVOT under the RV-PA conduit. < This report is a rare case of successful catheter ablation of unmappable ventricular tachycardia (VT) in a patient with complete transposition of the great arteries after Rastelli repair. The VT was unmappable because of intolerable hemodynamics. However, we could speculate the exit or isthmus of the VT by pace mapping or local potential and eliminate the VT.>.
先天性心脏病修复术后室性心动过速(VT)的导管消融(CA)可能具有挑战性,因为解剖结构复杂,且有时VT难以标测。在此,我们报告一名41岁女性,在接受Rastelli修复术后的大动脉完全转位患者中,成功对无法标测的VT进行了CA。当标测导管置于高位右心室流出道(RVOT)时,通过程序电刺激诱发临床VT。VT发作时,右心室(RV)-肺动脉(PA)管道下方高位前RVOT处的局部电位与QRS波起始时的电位相等。由于血流动力学恶化,VT无法标测。还尝试在主动脉瓣尖和左心室流出道(LVOT)进行起搏标测。在无冠瓣尖观察到窦性心律时的碎裂电位,该部位的起搏QRS形态与临床VT相似,起搏至QRS波起始的延迟为55毫秒。然而,由于导管操作困难,无法在LVOT进行标测。成功在无冠瓣尖和RV-PA管道下方的高位前RVOT施加射频能量。<本报告是Rastelli修复术后大动脉完全转位患者成功对无法标测的室性心动过速(VT)进行导管消融的罕见病例。VT因血流动力学难以耐受而无法标测。然而,我们可以通过起搏标测或局部电位推测VT的出口或峡部并消除VT。>