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本文引用的文献

1
Safety of AADC Gene Therapy for Moderately Advanced Parkinson Disease: Three-Year Outcomes From the PD-1101 Trial.AADC 基因治疗中度晚期帕金森病的安全性:PD-1101 试验的 3 年结果。
Neurology. 2022 Jan 4;98(1):e40-e50. doi: 10.1212/WNL.0000000000012952. Epub 2021 Oct 14.
2
Unregulated cytosolic dopamine causes neurodegeneration associated with oxidative stress in mice.未受调控的胞质多巴胺会导致与小鼠氧化应激相关的神经退行性变。
J Neurosci. 2008 Jan 9;28(2):425-33. doi: 10.1523/JNEUROSCI.3602-07.2008.
3
Vesicular monoamine transporter-2 and aromatic L-amino acid decarboxylase enhance dopamine delivery after L-3, 4-dihydroxyphenylalanine administration in Parkinsonian rats.在帕金森病大鼠中,囊泡单胺转运体-2和芳香族L-氨基酸脱羧酶可增强左旋多巴给药后的多巴胺递送。
J Neurosci. 1999 Apr 15;19(8):3266-74. doi: 10.1523/JNEUROSCI.19-08-03266.1999.
4
The role of glutathione in dopaminergic neuronal survival.谷胱甘肽在多巴胺能神经元存活中的作用。
J Neurochem. 1997 Nov;69(5):1850-8. doi: 10.1046/j.1471-4159.1997.69051850.x.
5
Regulation of dopamine production by genetically modified primary fibroblasts.转基因原代成纤维细胞对多巴胺生成的调控
J Neurosci. 1993 Dec;13(12):5203-11. doi: 10.1523/JNEUROSCI.13-12-05203.1993.

Reader Response: Safety of AADC Gene Therapy for Moderately Advanced Parkinson Disease: Three-Year Outcomes From the PD-1101 Trial.

作者信息

Kang Un Jung, Nakamura Ken, Zhuang Xiaoxi

机构信息

(New York).

(San Francisco).

出版信息

Neurology. 2022 Aug 9;99(6):258-259. doi: 10.1212/WNL.0000000000201002.

DOI:10.1212/WNL.0000000000201002
PMID:35940895
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10499428/
Abstract
摘要