Correction: Thalidomide with blockade of costimulatory molecules prolongs the survival of alloantigen-primed mice with cardiac allografts.
作者信息
Zhu Maoshu, Ma Yunhan, Tan Kai, Zhang Liyi, Wang Zhaowei, Li Yongsheng, Chen Yingyu, Guo Junjun, Yan Guoliang, Qi Zhongquan
机构信息
Xiang'an Branch, The First Affiliated Hospital of Xiamen University, Xiamen, 361100, Fujian, China.
The Fifth Hospital of Xiamen, Xiamen, 361100, Fujian, China.
出版信息
BMC Immunol. 2022 Aug 9;23(1):38. doi: 10.1186/s12865-022-00512-5.
Abstract
摘要
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Correction: Thalidomide with blockade of costimulatory molecules prolongs the survival of alloantigen-primed mice with cardiac allografts.更正：沙利度胺联合共刺激分子阻断可延长接受心脏同种异体移植的同种抗原致敏小鼠的生存期。
BMC Immunol. 2022 Aug 9;23(1):38. doi: 10.1186/s12865-022-00512-5.
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Thalidomide with blockade of co-stimulatory molecules prolongs the survival of alloantigen-primed mice with cardiac allografts.沙利度胺联合共刺激分子阻断延长致敏小鼠心脏移植存活。
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Monoclonal antibody treatment to prolong the secondary cardiac allograft survival in alloantigen-primed mice.单克隆抗体治疗延长同种抗原致敏小鼠的次级心脏移植物存活。
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Costimulatory blockade prevents early rejection, promotes lymphocyte apoptosis, and inhibits the upregulation of intragraft interleukin-6 in an orthotopic liver transplant model in the rat.共刺激阻断可预防大鼠原位肝移植模型中的早期排斥反应，促进淋巴细胞凋亡，并抑制移植肝内白细胞介素-6的上调。
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本文引用的文献
1
Thalidomide with blockade of co-stimulatory molecules prolongs the survival of alloantigen-primed mice with cardiac allografts.沙利度胺联合共刺激分子阻断延长致敏小鼠心脏移植存活。
BMC Immunol. 2020 Apr 16;21(1):19. doi: 10.1186/s12865-020-00352-1.
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