Correction: ELAVL1 is transcriptionally activated by FOXC1 and promotes ferroptosis in myocardial ischemia/reperfusion injury by regulating autophagy.
作者信息
Chen Hui-Yong, Xiao Ze-Zhou, Ling Xiao, Xu Rong-Ning, Zhu Peng, Zheng Shao-Yi
机构信息
Department of Cardiovascular Surgery, Nanfang Hospital, Southern Medical University, No. 1838 North Guangzhou Avenue, Baiyun District, Guangzhou, 510515, Guangdong, People's Republic of China.
Department of Thoracic Surgery, Yuebei People's Hospital, Shantou University, Shaoguan, 512026, Guangdong, People's Republic of China.
出版信息
Mol Med. 2022 Aug 15;28(1):96. doi: 10.1186/s10020-022-00529-x.
Abstract
摘要
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Correction: ELAVL1 is transcriptionally activated by FOXC1 and promotes ferroptosis in myocardial ischemia/reperfusion injury by regulating autophagy.更正：ELAVL1由FOXC1转录激活，并通过调节自噬促进心肌缺血/再灌注损伤中的铁死亡。
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本文引用的文献
1
ELAVL1 is transcriptionally activated by FOXC1 and promotes ferroptosis in myocardial ischemia/reperfusion injury by regulating autophagy.ELAVL1 通过 FOXC1 转录激活，并通过调节自噬促进心肌缺血/再灌注损伤中的铁死亡。
Mol Med. 2021 Feb 10;27(1):14. doi: 10.1186/s10020-021-00271-w.
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