• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

Retraction Note: Isovitexin reduces carcinogenicity and stemness in hepatic carcinoma stem-like cells by modulating MnSOD and FoxM1.

作者信息

Cao Xiaocheng, Liu Lihua, Yuan Qing, Li Xiang, Cui Yinghong, Ren Kaiqun, Zou Chang, Chen A, Xu Chang, Qiu Yebei, Quan Meifang, Zhang Jiansong, Cao Jianguo, Chen Xiangding

机构信息

Department of Pharmaceutical Science, Medical College, Hunan Normal University, Changsha, 410013, Hunan, China.

Key Laboratory of Study and Discover of Small Targeted Molecules of Hunan Province, Changsha, 410013, Hunan, China.

出版信息

J Exp Clin Cancer Res. 2022 Aug 30;41(1):263. doi: 10.1186/s13046-022-02470-7.

DOI:10.1186/s13046-022-02470-7
PMID:36038923
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9426262/
Abstract
摘要

相似文献

1
Retraction Note: Isovitexin reduces carcinogenicity and stemness in hepatic carcinoma stem-like cells by modulating MnSOD and FoxM1.撤稿声明:异荭草素通过调节锰超氧化物歧化酶和叉头框蛋白M1降低肝癌干细胞样细胞的致癌性和干性。
J Exp Clin Cancer Res. 2022 Aug 30;41(1):263. doi: 10.1186/s13046-022-02470-7.
2
Isovitexin reduces carcinogenicity and stemness in hepatic carcinoma stem-like cells by modulating MnSOD and FoxM1.异牡荆黄素通过调节 MnSOD 和 FoxM1 降低肝癌干细胞的致癌性和干性。
J Exp Clin Cancer Res. 2019 Jun 17;38(1):264. doi: 10.1186/s13046-019-1244-6.
3
Modulation of MnSOD and FoxM1 Is Involved in Invasion and EMT Suppression by Isovitexin in Hepatocellular Carcinoma Cells.异荭草素对锰超氧化物歧化酶和叉头框蛋白M1的调节参与其对肝癌细胞侵袭和上皮-间质转化的抑制作用。
Cancer Manag Res. 2020 Jul 14;12:5759-5771. doi: 10.2147/CMAR.S245283. eCollection 2020.
4
Modulation of MnSOD and FoxM1 is Involved in Invasion and EMT Suppression by Isovitexin in Hepatocellular Carcinoma Cells [Retraction].异荭草素对锰超氧化物歧化酶和叉头框蛋白M1的调节参与其对肝癌细胞侵袭和上皮-间质转化的抑制作用[撤回声明]
Cancer Manag Res. 2022 Apr 19;14:1473-1474. doi: 10.2147/CMAR.S370617. eCollection 2022.
5
Upregulation of FoxM1 by MnSOD Overexpression Contributes to Cancer Stem-Like Cell Characteristics in the Lung Cancer H460 Cell Line.超表达锰超氧化物歧化酶对FoxM1的上调作用促使肺癌H460细胞系具有癌症干细胞样特征。
Technol Cancer Res Treat. 2018 Jan 1;17:1533033818789635. doi: 10.1177/1533033818789635.
6
Retracted: Isovitexin Suppresses Stemness of Lung Cancer Stem-Like Cells through Blockage of MnSOD/CaMKII/AMPK Signaling and Glycolysis Inhibition.撤回:异荭草素通过阻断锰超氧化物歧化酶/钙调蛋白激酶II/腺苷酸活化蛋白激酶信号通路和抑制糖酵解来抑制肺癌干细胞样细胞的干性。
Biomed Res Int. 2024 Jan 9;2024:9802384. doi: 10.1155/2024/9802384. eCollection 2024.
7
MnSOD promotes tumor invasion via upregulation of FoxM1-MMP2 axis and related with poor survival and relapse in lung adenocarcinomas.MnSOD 通过上调 FoxM1-MMP2 轴促进肿瘤侵袭,与肺腺癌患者不良生存和复发相关。
Mol Cancer Res. 2013 Mar;11(3):261-71. doi: 10.1158/1541-7786.MCR-12-0527. Epub 2012 Dec 27.
8
The DNMT1/miR-34a/FOXM1 Axis Contributes to Stemness of Liver Cancer Cells.DNMT1/miR-34a/FOXM1轴促成肝癌细胞的干性。
J Oncol. 2020 Apr 5;2020:8978930. doi: 10.1155/2020/8978930. eCollection 2020.
9
A multi-mode Wnt- and stemness-regulatory module dictated by FOXM1 and ASPM isoform I in gastric cancer.FOXM1 和 ASPM 异构体 I 调控的多模式 Wnt 和干性调控模块在胃癌中的作用。
Gastric Cancer. 2021 May;24(3):624-639. doi: 10.1007/s10120-020-01154-5. Epub 2021 Jan 29.
10
Retraction Note: 8-bromo-7-methoxychrysin suppress stemness of SMMC-7721 cells induced by co-culture of liver cancer stem-like cells with hepatic stellate cells.撤稿说明:8-溴-7-甲氧基白杨素抑制肝癌干细胞样细胞与肝星状细胞共培养诱导的SMMC-7721细胞干性。
BMC Cancer. 2023 Dec 7;23(1):1203. doi: 10.1186/s12885-023-11698-1.

本文引用的文献

1
Genistein inhibits stemness of SKOV3 cells induced by macrophages co-cultured with ovarian cancer stem-like cells through IL-8/STAT3 axis.金雀异黄素通过 IL-8/STAT3 轴抑制巨噬细胞与卵巢癌干细胞样细胞共培养诱导的 SKOV3 细胞干性。
J Exp Clin Cancer Res. 2019 Jan 15;38(1):19. doi: 10.1186/s13046-018-1010-1.