uEVs: A Potential Tool for Examining Renal Epithelial Cells.
作者信息
Deck Katherine S, Mu Shengyu
机构信息
Department of Pharmacology and Toxicology, University of Arkansas for Medical Sciences, Little Rock, Arkansas.
出版信息
Kidney360. 2022 Apr 26;3(5):796-798. doi: 10.34067/KID.0001832022. eCollection 2022 May 26.
Abstract
摘要
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本文引用的文献
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Acute Intravenous NaCl and Volume Expansion Reduces Sodium-Chloride Cotransporter Abundance and Phosphorylation in Urinary Extracellular Vesicles.急性静脉内 NaCl 和容量扩张减少尿细胞外囊泡中钠-氯共转运体的丰度和磷酸化。
Kidney360. 2022 Mar 1;3(5):910-921. doi: 10.34067/KID.0000362022. eCollection 2022 May 26.
2
Large-Scale Proteomic Assessment of Urinary Extracellular Vesicles Highlights Their Reliability in Reflecting Protein Changes in the Kidney.大规模蛋白质组学分析尿液细胞外囊泡，突出其在反映肾脏蛋白质变化方面的可靠性。
J Am Soc Nephrol. 2021 Sep;32(9):2195-2209. doi: 10.1681/ASN.2020071035. Epub 2021 Jul 6.
3
Proteomic Profile of Urinary Extracellular Vesicles Identifies AGP1 as a Potential Biomarker of Primary Aldosteronism.尿液细胞外囊泡的蛋白质组学特征鉴定出AGP1作为原发性醛固酮增多症的潜在生物标志物。
Endocrinology. 2021 Apr 1;162(4). doi: 10.1210/endocr/bqab032.
4
Extracellular Vesicles in Essential Hypertension: Hidden Messengers.原发性高血压中的细胞外囊泡：隐匿的信使。
Curr Hypertens Rep. 2020 Sep 3;22(10):76. doi: 10.1007/s11906-020-01084-8.
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Regulation of the Renal NaCl Cotransporter and Its Role in Potassium Homeostasis.肾脏 NaCl 共转运蛋白的调节及其在钾稳态中的作用。
Physiol Rev. 2020 Jan 1;100(1):321-356. doi: 10.1152/physrev.00044.2018.
6
In human nephrectomy specimens, the kidney level of tubular transport proteins does not correlate with their abundance in urinary extracellular vesicles.在人类肾切除术标本中，管状转运蛋白的肾脏水平与其在尿细胞外囊泡中的丰度不相关。
Am J Physiol Renal Physiol. 2019 Sep 1;317(3):F560-F571. doi: 10.1152/ajprenal.00242.2019. Epub 2019 Jun 26.
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Characterization and Gene Expression Analysis of Serum-Derived Extracellular Vesicles in Primary Aldosteronism.原发性醛固酮增多症患者血清衍生细胞外囊泡的特征和基因表达分析。
Hypertension. 2019 Aug;74(2):359-367. doi: 10.1161/HYPERTENSIONAHA.119.12944. Epub 2019 Jun 24.
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Activation of the Endogenous Renin-Angiotensin-Aldosterone System or Aldosterone Administration Increases Urinary Exosomal Sodium Channel Excretion.内源性肾素-血管紧张素-醛固酮系统的激活或醛固酮给药会增加尿外泌体钠通道排泄。
J Am Soc Nephrol. 2016 Feb;27(2):646-56. doi: 10.1681/ASN.2014111137. Epub 2015 Jun 25.
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Biological properties of extracellular vesicles and their physiological functions.细胞外囊泡的生物学特性及其生理功能。
J Extracell Vesicles. 2015 May 14;4:27066. doi: 10.3402/jev.v4.27066. eCollection 2015.
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Are sodium transporters in urinary exosomes reliable markers of tubular sodium reabsorption in hypertensive patients?在高血压患者中，尿外泌体中的钠转运体是否是可靠的肾小管钠重吸收标志物？
Nephron Physiol. 2010;114(3):p25-34. doi: 10.1159/000274468. Epub 2010 Jan 12.
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