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缺乏亲环素 D 的小鼠肝和血浆脂质组的重塑。

Remodeling of Liver and Plasma Lipidomes in Mice Lacking Cyclophilin D.

机构信息

Department of Biochemistry and Medical Chemistry, Medical School, University of Pecs, 7624 Pecs, Hungary.

Institute of Biochemistry, Biological Research Centre, Eötvös Loránd Research Network, 6726 Szeged, Hungary.

出版信息

Int J Mol Sci. 2022 Sep 24;23(19):11274. doi: 10.3390/ijms231911274.

DOI:10.3390/ijms231911274
PMID:36232575
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9569465/
Abstract

In recent years, several studies aimed to investigate the metabolic effects of non-functioning or absent cyclophilin D (CypD), a crucial regulatory component of mitochondrial permeability transition pores. It has been reported that the lack of CypD affects glucose and lipid metabolism. However, the findings are controversial regarding the metabolic pathways involved, and most reports describe the effect of a high-fat diet on metabolism. We performed a lipidomic analysis of plasma and liver samples of CypD-/- and wild-type (WT) mice to reveal the lipid-specific alterations resulting from the absence of CypD. In the CypD-/- mice compared to the WT animals, we found a significant change in 52% and 47% of the measured 225 and 201 lipid species in liver and plasma samples, respectively. The higher total lipid content detected in these tissues was not accompanied by abdominal fat accumulation assessed by nuclear magnetic resonance imaging. We also documented characteristic changes in the lipid composition of the liver and plasma as a result of CypD ablation with the relative increase in polyunsaturated membrane lipid species. In addition, we did not observe remarkable differences in the lipid distribution of hepatocytes using histochemistry, but we found characteristic changes in the hepatocyte ultrastructure in CypD-/- animals using electron microscopy. Our results highlight the possible long-term effects of CypD inhibition as a novel therapeutic consideration for various diseases.

摘要

近年来,有几项研究旨在探讨无功能或缺失的亲环素 D(CypD)对线粒体通透性转换孔的代谢影响,CypD 是线粒体通透性转换孔的关键调节组成部分。据报道,CypD 的缺失会影响葡萄糖和脂质代谢。然而,关于涉及的代谢途径的研究结果存在争议,并且大多数报道都描述了高脂肪饮食对代谢的影响。我们对 CypD-/-和野生型(WT)小鼠的血浆和肝脏样本进行了脂质组学分析,以揭示 CypD 缺失导致的脂质特异性变化。与 WT 动物相比,我们在 CypD-/-小鼠的肝脏和血浆样本中分别发现了 52%和 47%的 225 种和 201 种测量脂质中存在显著变化。这些组织中检测到的总脂质含量增加,但并未伴有通过核磁共振成像评估的腹部脂肪堆积。我们还记录了 CypD 缺失导致的肝脏和血浆脂质组成的特征变化,表现为多不饱和膜脂质的相对增加。此外,我们通过组织化学未观察到肝细胞中脂质分布的显著差异,但通过电子显微镜发现 CypD-/-动物的肝细胞超微结构存在特征性变化。我们的研究结果强调了 CypD 抑制作为各种疾病的新型治疗考虑因素的可能长期影响。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/74ac/9569465/48be25da0db7/ijms-23-11274-g006.jpg
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本文引用的文献

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Biochem Biophys Res Commun. 2022 Feb 26;594:168-176. doi: 10.1016/j.bbrc.2022.01.059. Epub 2022 Jan 18.
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Targeting the mitochondrial permeability transition pore for drug discovery: Challenges and opportunities.靶向线粒体通透性转换孔的药物发现:挑战与机遇。
Mitochondrion. 2022 Mar;63:57-71. doi: 10.1016/j.mito.2022.01.006. Epub 2022 Jan 22.
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The mitochondrial permeability transition: Recent progress and open questions.
线粒体通透性转换:最新进展与未解问题
FEBS J. 2022 Nov;289(22):7051-7074. doi: 10.1111/febs.16254. Epub 2021 Nov 12.
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Male and Female Animals Respond Differently to High-Fat Diet and Regular Exercise Training in a Mouse Model of Hyperlipidemia.高脂饮食和规律运动训练在高脂血症小鼠模型中对雌雄动物的影响不同。
Int J Mol Sci. 2021 Apr 18;22(8):4198. doi: 10.3390/ijms22084198.
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Cyclophilin D: An Integrator of Mitochondrial Function.亲环素D:线粒体功能的整合者
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Targeting putative components of the mitochondrial permeability transition pore for novel therapeutics.针对线粒体通透性转换孔的假定成分进行新型治疗。
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