Correction: ICAM-1 promotes cancer progression by regulating SRC activity as an adapter protein in colorectal cancer.
作者信息
Lim Eun-Ji, Kang Jae-Hyeok, Kim Yeon-Ju, Kim Seungmo, Lee Su-Jae
机构信息
Department of Life Science, Research Institute for Natural Sciences, Hanyang University, Seoul, 04763, Korea.
出版信息
Cell Death Dis. 2022 Nov 18;13(11):968. doi: 10.1038/s41419-022-05425-0.
Abstract
摘要
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Correction: ICAM-1 promotes cancer progression by regulating SRC activity as an adapter protein in colorectal cancer.更正：在结直肠癌中，细胞间黏附分子-1（ICAM-1）作为衔接蛋白通过调节Src活性促进癌症进展。
Cell Death Dis. 2022 Nov 18;13(11):968. doi: 10.1038/s41419-022-05425-0.
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ICAM-1 promotes cancer progression by regulating SRC activity as an adapter protein in colorectal cancer.细胞间黏附分子-1 通过作为结直肠癌细胞中的衔接蛋白调节 SRC 活性来促进癌症进展。
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SNX10 (sorting nexin 10) inhibits colorectal cancer initiation and progression by controlling autophagic degradation of SRC.分选连接蛋白 10（sorting nexin 10）通过控制 SRC 的自噬降解来抑制结直肠癌细胞的发生和发展。
Autophagy. 2020 Apr;16(4):735-749. doi: 10.1080/15548627.2019.1632122. Epub 2019 Jul 4.
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Interferon-gamma-induced epithelial ICAM-1 expression and monocyte adhesion. Involvement of protein kinase C-dependent c-Src tyrosine kinase activation pathway.干扰素-γ诱导上皮细胞细胞间黏附分子-1表达及单核细胞黏附。蛋白激酶C依赖性c-Src酪氨酸激酶激活途径的参与。
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Activation of SRC tyrosine kinases in response to ICAM-1 ligation in pulmonary microvascular endothelial cells.肺微血管内皮细胞中，SRC酪氨酸激酶因细胞间黏附分子-1（ICAM-1）连接而被激活。
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Endothelial cell SHP-2 negatively regulates neutrophil adhesion and promotes transmigration by enhancing ICAM-1-VE-cadherin interaction.内皮细胞中的SHP-2通过增强ICAM-1与VE-钙黏蛋白的相互作用，对中性粒细胞黏附起负向调节作用，并促进其迁移。
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