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Corrigendum: A novel N-arylpyridone compound alleviates the inflammatory and fibrotic reaction of silicosis by inhibiting the ASK1-p38 pathway and regulating macrophage polarization.勘误：一种新型N-芳基吡啶酮化合物通过抑制ASK1-p38途径和调节巨噬细胞极化减轻矽肺的炎症和纤维化反应。
Front Pharmacol. 2023 Jan 18;13:1109002. doi: 10.3389/fphar.2022.1109002. eCollection 2022.
2
Erratum: Addendum: A novel N-Arylpyridone compound alleviates the inflammatory and fibrotic reaction of silicosis by inhibiting the ASK1-p38 pathway and regulating macrophage polarization.勘误：补遗：一种新型N-芳基吡啶酮化合物通过抑制ASK1-p38通路和调节巨噬细胞极化减轻矽肺的炎症和纤维化反应。
Front Pharmacol. 2023 Jan 18;13:1108989. doi: 10.3389/fphar.2022.1108989. eCollection 2022.
3
A Novel N-Arylpyridone Compound Alleviates the Inflammatory and Fibrotic Reaction of Silicosis by Inhibiting the ASK1-p38 Pathway and Regulating Macrophage Polarization.一种新型N-芳基吡啶酮化合物通过抑制ASK1-p38信号通路和调节巨噬细胞极化减轻矽肺的炎症和纤维化反应。
Front Pharmacol. 2022 Mar 23;13:848435. doi: 10.3389/fphar.2022.848435. eCollection 2022.
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Pirfenidone ameliorates pulmonary inflammation and fibrosis in a rat silicosis model by inhibiting macrophage polarization and JAK2/STAT3 signaling pathways.吡非尼酮通过抑制巨噬细胞极化和 JAK2/STAT3 信号通路改善大鼠矽肺模型中的肺炎症和纤维化。
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Silica particles disorganize the polarization of pulmonary macrophages in mice.二氧化硅颗粒使小鼠肺部巨噬细胞的极化紊乱。
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Novel ASK1 Inhibitor AGI-1067 Attenuates AGE-Induced Fibrotic Response by Suppressing the MKKs/p38 MAPK Pathway in Human Coronary Arterial Smooth Muscle Cells.新型ASK1抑制剂AGI-1067通过抑制人冠状动脉平滑肌细胞中的MKKs/p38丝裂原活化蛋白激酶途径减轻晚期糖基化终产物诱导的纤维化反应。
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Corrigendum: Deferoxamine alleviates osteoarthritis by inhibiting chondrocyte ferroptosis and activating the Nrf2 pathway.勘误：去铁胺通过抑制软骨细胞铁死亡和激活Nrf2途径减轻骨关节炎。
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VX-765 attenuates silica-induced lung inflammatory injury and fibrosis by modulating alveolar macrophages pyroptosis in mice.VX-765通过调节小鼠肺泡巨噬细胞焦亡减轻二氧化硅诱导的肺部炎性损伤和纤维化。
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Regulation of Microglia and Macrophage Polarization via Apoptosis Signal-Regulating Kinase 1 Silencing after Ischemic/Hypoxic Injury.缺血/缺氧损伤后通过沉默凋亡信号调节激酶1调控小胶质细胞和巨噬细胞极化
Front Mol Neurosci. 2017 Aug 14;10:261. doi: 10.3389/fnmol.2017.00261. eCollection 2017.

勘误：一种新型N-芳基吡啶酮化合物通过抑制ASK1-p38途径和调节巨噬细胞极化减轻矽肺的炎症和纤维化反应。

Corrigendum: A novel N-arylpyridone compound alleviates the inflammatory and fibrotic reaction of silicosis by inhibiting the ASK1-p38 pathway and regulating macrophage polarization.

作者信息

Fan Mingming, Xiao Huijuan, Song Dingyun, Zhu Lili, Zhang Jie, Zhang Xinran, Wang Jing, Dai Huaping, Wang Chen

机构信息

Department of Respiratory Medicine, The Second Hospital of Jilin University, Jilin, China.

Department of Pulmonary and Critical Care Medicine Center of Respiratory Medicine, China-Japan Friendship Hospital, Capital Medical University, National Center for Respiratory Medicine, National Clinical Research Center for Respiratory Diseases, Institute of Respiratory Medicine, Chinese Academy of Medical Sciences, Peking Union Medical College, Beijing, China.

出版信息

Front Pharmacol. 2023 Jan 18;13:1109002. doi: 10.3389/fphar.2022.1109002. eCollection 2022.

DOI:10.3389/fphar.2022.1109002
PMID:36744209
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9890144/
Abstract

[This corrects the article DOI: 10.3389/fphar.2022.848435.].

摘要

[本文更正了文章的数字对象标识符：10.3389/fphar.2022.848435。]