Calcium "stress" adds a third hit in driving heart failure with preserved ejection fraction.

作者信息

Fujita Kyohei, Ellis Matthew W, Sheikh Farah

机构信息

Department of Medicine, University of California San Diego, La Jolla, California, United States.

出版信息

Am J Physiol Heart Circ Physiol. 2023 Apr 1;324(4):H414-H416. doi: 10.1152/ajpheart.00075.2023. Epub 2023 Feb 10.

DOI:10.1152/ajpheart.00075.2023

PMID:36763507

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9988519/

Abstract

摘要

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[2]

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[5]

Nitrosative stress drives heart failure with preserved ejection fraction.

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[6]

Histone deacetylase activity governs diastolic dysfunction through a nongenomic mechanism.

Sci Transl Med. 2018-2-7

[7]

Epidemiology of heart failure with preserved ejection fraction.

Nat Rev Cardiol. 2017-5-11

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Histone deacetylase inhibition blunts ischemia/reperfusion injury by inducing cardiomyocyte autophagy.

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Calcium "stress" adds a third hit in driving heart failure with preserved ejection fraction.

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