PCSK6: The Endogenous PAR-1 Agonist Driving Pulmonary Fibrosis?
作者信息
Spek C Arnold, Duitman JanWillem
机构信息
Center for Experimental and Molecular Medicine.
Cancer Biology and Immunology, Cancer Center Amsterdam, Amsterdam, the Netherlands; and.
出版信息
Am J Respir Crit Care Med. 2023 Jun 15;207(12):1643-1644. doi: 10.1164/rccm.202303-0355LE.
Abstract
摘要
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本文引用的文献
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PCSK6 and Survival in Idiopathic Pulmonary Fibrosis.载脂蛋白 C3 与特发性肺纤维化患者的生存。
Am J Respir Crit Care Med. 2023 Jun 1;207(11):1515-1524. doi: 10.1164/rccm.202205-0845OC.
2
Vorapaxar proven to be a promising candidate for pulmonary fibrosis by intervening in the PAR1/JAK2/STAT1/3 signaling pathway-an experimental in vitro and vivo study.通过干预PAR1/JAK2/STAT1/3信号通路，沃拉帕沙被证明是肺纤维化的一个有前景的候选药物——一项体外和体内实验研究。
Eur J Pharmacol. 2023 Mar 15;943:175438. doi: 10.1016/j.ejphar.2022.175438. Epub 2023 Jan 20.
3
Protease-Activated Receptor Antagonist for Reducing Cardiovascular Events - A Review on Vorapaxar.用于减少心血管事件的蛋白酶激活受体拮抗剂——沃拉帕沙综述
Curr Probl Cardiol. 2023 Mar;48(3):101035. doi: 10.1016/j.cpcardiol.2021.101035. Epub 2021 Oct 28.
4
Unfavourable effects of medically indicated oral anticoagulants on survival in idiopathic pulmonary fibrosis.医学指征地口服抗凝剂对特发性肺纤维化患者生存的不利影响。
Eur Respir J. 2016 Jun;47(6):1776-84. doi: 10.1183/13993003.02087-2015. Epub 2016 Apr 21.
5
Neuroinflammation-Induced Interactions between Protease-Activated Receptor 1 and Proprotein Convertases in HIV-Associated Neurocognitive Disorder.神经炎症诱导的蛋白酶激活受体1与前蛋白转化酶在HIV相关神经认知障碍中的相互作用
Mol Cell Biol. 2015 Nov;35(21):3684-700. doi: 10.1128/MCB.00764-15. Epub 2015 Aug 17.
6
Targeting protease activated receptor-1 with P1pal-12 limits bleomycin-induced pulmonary fibrosis.靶向蛋白酶激活受体-1 用 P1pal-12 限制博来霉素诱导的肺纤维化。
Thorax. 2014 Feb;69(2):152-60. doi: 10.1136/thoraxjnl-2013-203877. Epub 2013 Sep 12.
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