勘误：对三阴性乳腺癌具有选择性活性的Wnt信号通路抑制剂：从噻吩并嘧啶到喹唑啉抑制剂。
Erratum: Wnt-pathway inhibitors with selective activity against triple-negative breast cancer: From thienopyrimidine to quinazoline inhibitors.
机构信息
Frontiers Media SA, Lausanne, Switzerland.
出版信息
Front Pharmacol. 2023 Mar 7;14:1173490. doi: 10.3389/fphar.2023.1173490. eCollection 2023.
Abstract
[This corrects the article DOI: 10.3389/fphar.2022.1045102.].
摘要
[本文更正了文章的数字对象标识符：10.3389/fphar.2022.1045102。]
相似文献
1
Erratum: Wnt-pathway inhibitors with selective activity against triple-negative breast cancer: From thienopyrimidine to quinazoline inhibitors.勘误：对三阴性乳腺癌具有选择性活性的Wnt信号通路抑制剂：从噻吩并嘧啶到喹唑啉抑制剂。
Front Pharmacol. 2023 Mar 7;14:1173490. doi: 10.3389/fphar.2023.1173490. eCollection 2023.
2
Wnt-pathway inhibitors with selective activity against triple-negative breast cancer: From thienopyrimidine to quinazoline inhibitors.对三阴性乳腺癌具有选择性活性的Wnt信号通路抑制剂：从噻吩并嘧啶到喹唑啉抑制剂
Front Pharmacol. 2022 Oct 28;13:1045102. doi: 10.3389/fphar.2022.1045102. eCollection 2022.
3
Corrigendum: AFAP1-AS1 Promotes Epithelial-Mesenchymal Transition and Tumorigenesis Through Wnt/β-Catenin Signaling Pathway in Triple-Negative Breast Cancer.勘误：AFAP1-AS1通过Wnt/β-连环蛋白信号通路促进三阴性乳腺癌的上皮-间质转化和肿瘤发生。
Front Pharmacol. 2020 Jul 20;11:1107. doi: 10.3389/fphar.2020.01107. eCollection 2020.
4
Targeting Wnt/β-catenin signaling pathway in triple-negative breast cancer by benzylic organotrisulfides: Contribution of the released hydrogen sulfide towards potent anti-cancer activity.通过苄基有机三硫化物靶向三阴性乳腺癌中的 Wnt/β-连环蛋白信号通路：释放的硫化氢对有效抗癌活性的贡献。
Free Radic Biol Med. 2022 Oct;191:82-96. doi: 10.1016/j.freeradbiomed.2022.08.029. Epub 2022 Aug 28.
5
Identification of quinazoline compounds as novel potent inhibitors of Wnt/β-catenin signaling in colorectal cancer cells.喹唑啉化合物作为结直肠癌细胞中Wnt/β-连环蛋白信号通路新型强效抑制剂的鉴定。
Oncotarget. 2016 Mar 8;7(10):11263-70. doi: 10.18632/oncotarget.7019.
6
Preferential Inhibition of Wnt/β-Catenin Signaling by Novel Benzimidazole Compounds in Triple-Negative Breast Cancer.新型苯并咪唑类化合物对三阴性乳腺癌中 Wnt/β-连环蛋白信号的优先抑制作用。
Int J Mol Sci. 2018 May 20;19(5):1524. doi: 10.3390/ijms19051524.
7
[Molecular mechanisms of resistance to phosphatidyl inositol 3-kinase inhibitors in triple-negative breast cancer cells].三阴性乳腺癌细胞对磷脂酰肌醇3-激酶抑制剂耐药的分子机制
Zhonghua Zhong Liu Za Zhi. 2016 Aug;38(8):578-88. doi: 10.3760/cma.j.issn.0253-3766.2016.08.004.
8
Role of microRNA/lncRNA Intertwined With the Wnt/β-Catenin Axis in Regulating the Pathogenesis of Triple-Negative Breast Cancer.微小RNA/长链非编码RNA与Wnt/β-连环蛋白轴相互交织在调节三阴性乳腺癌发病机制中的作用
Front Pharmacol. 2022 Jun 24;13:814971. doi: 10.3389/fphar.2022.814971. eCollection 2022.
9
CUEDC2 Drives β-Catenin Nuclear Translocation and Promotes Triple-Negative Breast Cancer Tumorigenesis.CUEDC2 驱动β-连环蛋白核易位并促进三阴性乳腺癌肿瘤发生。
Cells. 2022 Sep 29;11(19):3067. doi: 10.3390/cells11193067.
10
Inhibition of endoplasmic reticulum stress alleviates triple-negative breast cancer cell viability, migration, and invasion by Syntenin/SOX4/Wnt/β-catenin pathway via regulation of heat shock protein A4.抑制内质网应激通过调控热休克蛋白 A4 抑制 Syntenin/SOX4/Wnt/β-catenin 通路减轻三阴性乳腺癌细胞活力、迁移和侵袭。
Bioengineered. 2022 Apr;13(4):10564-10577. doi: 10.1080/21655979.2022.2062990.
Zotero 插件