[Acute respiratory insufficiency syndrome ("shock lung") in adults].

The causes of the development of acute respiratory insufficiency ("shock lung") may include any shock, burn disease, posttransfusion syndrome, hyperoxygenation and trauma of the lungs. Morphologically, against the background of inflation of the lung parenchyma, bilateral fine-focus atelectases, focal disorders of the circulation of the type of leukostases, sludge, leading to extensive hyalin (spodogenic) thromboses, develop. Secondarily, a small-focus edema, hemorrhages, sometimes, hyalin membranes, develop. Atelectases are initially reversible, but later, when surfactant desintegrates against the background of circulatory disorders, they become stable. The main mechanism of the development of this process consists in the release, during shock, of biologically active amines in the lungs leading to circulatory disorders in them. This is attested to by the possibility of producing the "shock lung" picture in intact animals by inoculation into their blood of plasma from the animals in a shock condition.