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Lipid catabolism and mitochondrial uncoupling are stimulated in brown adipose tissue of amyotrophic lateral sclerosis mouse models.

作者信息

Ciccarone Fabio, Castelli Serena, Lazzarino Giacomo, Scaricamazza Silvia, Mangione Renata, Bernardini Sergio, Apolloni Savina, D'Ambrosi Nadia, Ferri Alberto, Ciriolo Maria Rosa

机构信息

Department of Biology, University of Rome "Tor Vergata", 00133 Rome, Italy.

IRCCS San Raffaele Roma, 00166 Rome, Italy.

出版信息

Genes Dis. 2022 Apr 27;10(2):321-324. doi: 10.1016/j.gendis.2022.04.006. eCollection 2023 Mar.

DOI:10.1016/j.gendis.2022.04.006

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10201591/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c094/10201591/f153e761966b/gr1.jpg

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c094/10201591/f153e761966b/gr1.jpg

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c094/10201591/f153e761966b/gr1.jpg

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[1]

Lipid catabolism and mitochondrial uncoupling are stimulated in brown adipose tissue of amyotrophic lateral sclerosis mouse models.

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[3]

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[6]

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[7]

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Am J Physiol. 1990-2

[8]

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[9]

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[10]

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[2]

Brown Adipose Tissue undergoes pathological perturbations and shapes C2C12 myoblast homeostasis in the SOD1-G93A mouse model of Amyotrophic Lateral Sclerosis.

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[3]

Nutritional and Microbiota-Based Approaches in Amyotrophic Lateral Sclerosis: From Prevention to Treatment.

Nutrients. 2024-12-30

[4]

N-acetylaspartate promotes glycolytic-to-oxidative fiber-type switch and resistance to atrophic stimuli in myotubes.

Cell Death Dis. 2024-9-19

[5]

Evidence of Metabolic Dysfunction in Amyotrophic Lateral Sclerosis (ALS) Patients and Animal Models.

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本文引用的文献

[1]

Altered skeletal muscle glucose-fatty acid flux in amyotrophic lateral sclerosis.

Brain Commun. 2020-9-24

[2]

Skeletal-Muscle Metabolic Reprogramming in ALS-SOD1 Mice Predates Disease Onset and Is A Promising Therapeutic Target.

iScience. 2020-5-22

[3]

Anatomical, Physiological, and Functional Diversity of Adipose Tissue.

Cell Metab. 2018-1-9

[4]

Sensory and autonomic nervous system dysfunction in amyotrophic lateral sclerosis.

Neuropathol Appl Neurobiol. 2017-2

[5]

Energy metabolism in amyotrophic lateral sclerosis.

Lancet Neurol. 2010-10-27

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