CETAPS EA 3832, Faculty of Sport Sciences, University of Rouen, France.
CRIOBE UAR 3278, CNRS-EPHE-UPVD, Mooréa, Polynésie Française (French Polynesia).
Undersea Hyperb Med. 2023 Second Quarter;50(2):85-93. doi: 10.22462/01.01.2023.38.
During deep-sea freediving, many freedivers describe symptoms fairly similar to what has been related to inert gas narcosis in scuba divers. This manuscript aims to present the potential mechanisms underlying these symptoms. First, known mechanisms of narcosis are summarized while scuba diving. Then, potential underlying mechanisms involving the toxicity of gases (nitrogen, carbon dioxide and oxygen) are presented in freedivers. As the symptoms are felt during ascent, nitrogen is likely not the only gas involved. Since freedivers are frequently exposed to hypercapnic hypoxia toward the end of the dive, it is proposed that carbon dioxide and oxygen gases both play a major role. Finally, a new "hemodynamic hypothesis" based on the diving reflex is proposed in freedivers. The underlying mechanisms are undoubtedly multifactorial and therefore require further research and a new descriptive name. We propose a new term for these types of symptoms: freediving transient cognitive impairment.
在深海自由潜水过程中,许多自由潜水员描述的症状与水肺潜水员中与惰性气体麻醉相关的症状非常相似。本文旨在介绍这些症状背后的潜在机制。首先,总结了水肺潜水时麻醉的已知机制。然后,介绍了与气体(氮气、二氧化碳和氧气)毒性有关的潜在机制。由于症状是在上升过程中感觉到的,氮气可能不是唯一涉及的气体。由于自由潜水员在潜水结束时经常暴露于高碳酸血症性缺氧,因此认为二氧化碳和氧气都起主要作用。最后,在自由潜水员中提出了一种基于潜水反射的新的“血液动力学假说”。潜在机制无疑是多因素的,因此需要进一步的研究和一个新的描述性名称。我们为这些类型的症状提出了一个新的术语:自由潜水短暂认知障碍。