Scheie H G, Ewing M Q
Trans Ophthalmol Soc U K (1962). 1978 Apr;98(1):111-7.
The cause of aphakic glaucoma should always be determined because treatment must be directed towards the aetiological mechanism. We have defined early aphakic glaucoma as that occurring within the first 6 postoperative weeks and late aphakic glaucoma as occurring at any later time. Of the various causes of glaucoma in each category, among the most serious and challenging to treat are pupillary block and angle closure. These emanate from postoperative shallow or empty anterior chambers which are associated with wound fistula, choroidal detachment, or a combination of both. Other mechanisms producing aphakic glaucoma are inflammation, alpha chymotrypsin, epithelial downgrowth, and iris cysts. Pressure elevation unrelated to aphakic glaucoma may also result from intraocular tumours, recurrence of glaucoma secondary to uveitis, and undiagnosed preexisting primary glaucoma.
无晶状体性青光眼的病因必须始终明确,因为治疗必须针对病因机制。我们将早期无晶状体性青光眼定义为术后6周内发生的青光眼,晚期无晶状体性青光眼定义为在任何更晚时间发生的青光眼。在每一类青光眼中,各种病因中,最严重且治疗最具挑战性的是瞳孔阻滞和房角关闭。这些情况源于术后前房浅或无前房,这与伤口瘘、脉络膜脱离或两者兼有关。产生无晶状体性青光眼的其他机制包括炎症、α-糜蛋白酶、上皮植入和虹膜囊肿。与无晶状体性青光眼无关的眼压升高也可能由眼内肿瘤、葡萄膜炎继发青光眼复发以及未诊断出的既往原发性青光眼引起。