Reply to Matters Arising: Is congenital anosmia protective for Parkinson's disease triggered by pathogenic entrance through the nose?
作者信息
Arshamian Artin, Iravani Behzad, Lundström Johan N
机构信息
Department of Clinical Neuroscience, Karolinska Institutet, Stockholm, Sweden.
Department of Neurology and Neurological Science, Stanford University, Stanford, CA, USA.
出版信息
NPJ Parkinsons Dis. 2023 Jun 19;9(1):95. doi: 10.1038/s41531-023-00539-4.
Abstract
摘要
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Reply to Matters Arising: Is congenital anosmia protective for Parkinson's disease triggered by pathogenic entrance through the nose?对相关问题的回复：先天性嗅觉丧失对经鼻病原体入侵引发的帕金森病有保护作用吗？
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Matters Arising: Is congenital anosmia protective for Parkinson's disease triggered by pathogenic entrance through the nose?提出的问题：先天性嗅觉缺失对经鼻腔病原体侵入引发的帕金森病是否具有保护作用？
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本文引用的文献
1
Lifelong olfactory deprivation-dependent cortical reorganization restricted to orbitofrontal cortex.终身嗅觉剥夺依赖的皮质重组仅限于眶额皮质。
Hum Brain Mapp. 2023 Dec 15;44(18):6459-6470. doi: 10.1002/hbm.26522. Epub 2023 Nov 2.
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A postmortem study suggests a revision of the dual-hit hypothesis of Parkinson's disease.一项尸检研究表明需对帕金森病的双击假说进行修正。
NPJ Parkinsons Dis. 2022 Nov 30;8(1):166. doi: 10.1038/s41531-022-00436-2.
3
Is congenital anosmia protective for Parkinson's disease triggered by pathogenic entrance through the nose?先天性嗅觉缺失对经鼻腔致病入侵引发的帕金森病有保护作用吗？
NPJ Parkinsons Dis. 2022 Nov 10;8(1):152. doi: 10.1038/s41531-022-00425-5.
4
A non-invasive olfactory bulb measure dissociates Parkinson's patients from healthy controls and discloses disease duration.一种非侵入性嗅球测量方法可将帕金森病患者与健康对照区分开来，并揭示疾病持续时间。
NPJ Parkinsons Dis. 2021 Aug 18;7(1):75. doi: 10.1038/s41531-021-00220-8.
5
Ageing promotes pathological alpha-synuclein propagation and autonomic dysfunction in wild-type rats.衰老促进野生型大鼠病理性α-突触核蛋白的传播和自主神经功能障碍。
Brain. 2021 Jul 28;144(6):1853-1868. doi: 10.1093/brain/awab061.
6
Morphological changes in secondary, but not primary, sensory cortex in individuals with life-long olfactory sensory deprivation.嗅觉长期剥夺个体的次级而非初级感觉皮层的形态变化。
Neuroimage. 2020 Sep;218:117005. doi: 10.1016/j.neuroimage.2020.117005. Epub 2020 May 30.
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Non-invasive recording from the human olfactory bulb.从人类嗅球进行无创记录。
Nat Commun. 2020 Jan 31;11(1):648. doi: 10.1038/s41467-020-14520-9.
8
Human Olfaction without Apparent Olfactory Bulbs.人类嗅觉缺失而嗅球无明显异常
Neuron. 2020 Jan 8;105(1):35-45.e5. doi: 10.1016/j.neuron.2019.10.006. Epub 2019 Nov 6.
9
The olfactory bulb as the entry site for prion-like propagation in neurodegenerative diseases.嗅球作为神经退行性疾病中朊病毒样传播的进入部位。
Neurobiol Dis. 2018 Jan;109(Pt B):226-248. doi: 10.1016/j.nbd.2016.12.013. Epub 2016 Dec 20.
10
Intracerebral inoculation of pathological α-synuclein initiates a rapidly progressive neurodegenerative α-synucleinopathy in mice.脑内接种病理性 α-突触核蛋白会在小鼠中引发快速进行性神经退行性 α-突触核蛋白病。
J Exp Med. 2012 May 7;209(5):975-86. doi: 10.1084/jem.20112457. Epub 2012 Apr 16.
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