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Reply to Matters Arising: Is congenital anosmia protective for Parkinson's disease triggered by pathogenic entrance through the nose?对相关问题的回复：先天性嗅觉丧失对经鼻病原体入侵引发的帕金森病有保护作用吗？
NPJ Parkinsons Dis. 2023 Jun 19;9(1):95. doi: 10.1038/s41531-023-00539-4.
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本文引用的文献

1
Lifelong olfactory deprivation-dependent cortical reorganization restricted to orbitofrontal cortex.终身嗅觉剥夺依赖的皮质重组仅限于眶额皮质。
Hum Brain Mapp. 2023 Dec 15;44(18):6459-6470. doi: 10.1002/hbm.26522. Epub 2023 Nov 2.
2
A postmortem study suggests a revision of the dual-hit hypothesis of Parkinson's disease.一项尸检研究表明需对帕金森病的双击假说进行修正。
NPJ Parkinsons Dis. 2022 Nov 30;8(1):166. doi: 10.1038/s41531-022-00436-2.
3
Is congenital anosmia protective for Parkinson's disease triggered by pathogenic entrance through the nose?先天性嗅觉缺失对经鼻腔致病入侵引发的帕金森病有保护作用吗？
NPJ Parkinsons Dis. 2022 Nov 10;8(1):152. doi: 10.1038/s41531-022-00425-5.
4
A non-invasive olfactory bulb measure dissociates Parkinson's patients from healthy controls and discloses disease duration.一种非侵入性嗅球测量方法可将帕金森病患者与健康对照区分开来，并揭示疾病持续时间。
NPJ Parkinsons Dis. 2021 Aug 18;7(1):75. doi: 10.1038/s41531-021-00220-8.
5
Ageing promotes pathological alpha-synuclein propagation and autonomic dysfunction in wild-type rats.衰老促进野生型大鼠病理性α-突触核蛋白的传播和自主神经功能障碍。
Brain. 2021 Jul 28;144(6):1853-1868. doi: 10.1093/brain/awab061.
6
Morphological changes in secondary, but not primary, sensory cortex in individuals with life-long olfactory sensory deprivation.嗅觉长期剥夺个体的次级而非初级感觉皮层的形态变化。
Neuroimage. 2020 Sep;218:117005. doi: 10.1016/j.neuroimage.2020.117005. Epub 2020 May 30.
7
Non-invasive recording from the human olfactory bulb.从人类嗅球进行无创记录。
Nat Commun. 2020 Jan 31;11(1):648. doi: 10.1038/s41467-020-14520-9.
8
Human Olfaction without Apparent Olfactory Bulbs.人类嗅觉缺失而嗅球无明显异常
Neuron. 2020 Jan 8;105(1):35-45.e5. doi: 10.1016/j.neuron.2019.10.006. Epub 2019 Nov 6.
9
The olfactory bulb as the entry site for prion-like propagation in neurodegenerative diseases.嗅球作为神经退行性疾病中朊病毒样传播的进入部位。
Neurobiol Dis. 2018 Jan;109(Pt B):226-248. doi: 10.1016/j.nbd.2016.12.013. Epub 2016 Dec 20.
10
Intracerebral inoculation of pathological α-synuclein initiates a rapidly progressive neurodegenerative α-synucleinopathy in mice.脑内接种病理性 α-突触核蛋白会在小鼠中引发快速进行性神经退行性 α-突触核蛋白病。
J Exp Med. 2012 May 7;209(5):975-86. doi: 10.1084/jem.20112457. Epub 2012 Apr 16.

Reply to Matters Arising: Is congenital anosmia protective for Parkinson's disease triggered by pathogenic entrance through the nose?

作者信息

Arshamian Artin, Iravani Behzad, Lundström Johan N

机构信息

Department of Clinical Neuroscience, Karolinska Institutet, Stockholm, Sweden.

Department of Neurology and Neurological Science, Stanford University, Stanford, CA, USA.

出版信息

NPJ Parkinsons Dis. 2023 Jun 19;9(1):95. doi: 10.1038/s41531-023-00539-4.

DOI:10.1038/s41531-023-00539-4
PMID:37336882
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10279683/
Abstract
摘要