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去甲肾上腺素诱发的吉兰-巴雷综合征中的高血压

Norepinephrine-induced hypertension in Guillain Barré syndrome.

作者信息

Ventura H O, Messerli F H, Barron R E

出版信息

J Hypertens. 1986 Jun;4(3):265-7. doi: 10.1097/00004872-198606000-00002.

Abstract

Systemic haemodynamics, plasma catecholamine levels and diurnal variation of arterial pressure were studied in a 20-year-old patient with hypertension during Guillain Barré syndrome after complete resolution of the illness. Transient arterial hypertension during the course of Guillain Barré syndrome is characterized by an increased total peripheral resistance associated with elevated circulating norepinephrine levels, suggesting an over-activity of the sympathetic nervous system as the underlying mechanism of the elevated blood pressure.

摘要

在一名20岁患有吉兰-巴雷综合征的高血压患者疾病完全缓解后,对其全身血流动力学、血浆儿茶酚胺水平和动脉血压的昼夜变化进行了研究。吉兰-巴雷综合征病程中的短暂性动脉高血压的特征是总外周阻力增加,同时循环去甲肾上腺素水平升高,提示交感神经系统过度活跃是血压升高的潜在机制。

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