Oda H, Aizawa Y, Suzuki K, Sato M, Aizawa M, Murata M, Morikawa M, Otsuka H, Shibata A, Miyamura H
Jpn Circ J. 1986 Mar;50(3):283-7. doi: 10.1253/jcj.50.283.
A female patient had recurrent sustained ventricular tachycardia (VT) as a late sequela after complete repair of tetralogy of Fallot, and the clinically documented VT could be induced and terminated in a reproducible manner by programmed stimulation. VT was accompanied by fragmented electrical activities when the electrode was positioned at the outflow tract of the right ventricle where myotomy was added on the previous scar. The electrophysiological study revealed that the mechanism of VT was re-entry and that the focus of VT was located at the right ventricular outflow tract.
一名女性患者在法洛四联症完全修复术后出现复发性持续性室性心动过速（VT），作为晚期后遗症，经程序刺激可重复诱发出临床记录的VT并终止。当电极置于右心室流出道（先前瘢痕处进行了心肌切开术）时，VT伴有碎裂电活动。电生理研究表明，VT的机制是折返，且VT的起源位于右心室流出道。
