百草枯（1,1'-二甲基-4,4'-联吡啶鎓盐）毒理学综述。 - Suppr

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Review of the toxicology of paraquat (1,1'-dimethyl-4,4'-bipyridinium chloride).

作者信息

Haley T J

出版信息

Clin Toxicol. 1979;14(1):1-46. doi: 10.3109/15563657909030112.

DOI:10.3109/15563657909030112

PMID:373975

Abstract

摘要

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Review of the toxicology of paraquat (1,1'-dimethyl-4,4'-bipyridinium chloride).百草枯（1,1'-二甲基-4,4'-联吡啶鎓盐）毒理学综述。

Clin Toxicol. 1979;14(1):1-46. doi: 10.3109/15563657909030112.

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PARAQUAT TOLERANCE3 Is an E3 Ligase That Switches off Activated Oxidative Response by Targeting Histone-Modifying PROTEIN METHYLTRANSFERASE4b.百草枯耐受性3是一种E3连接酶，它通过靶向组蛋白修饰蛋白甲基转移酶4b来关闭激活的氧化应激反应。

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Paraquat Induces Apoptosis through a Mitochondria-Dependent Pathway in RAW264.7 Cells.百草枯通过线粒体依赖途径诱导RAW264.7细胞凋亡。

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Paraquat Resistant1, a Golgi-localized putative transporter protein, is involved in intracellular transport of paraquat.百草枯抗性 1 蛋白，一种定位于高尔基体的假定转运蛋白，参与百草枯的细胞内转运。

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Quantification of Paraquat, MPTP, and MPP+ in brain tissue using microwave-assisted solvent extraction (MASE) and high-performance liquid chromatography-mass spectrometry.使用微波辅助溶剂萃取（MASE）和高效液相色谱-质谱联用技术对脑组织中的百草枯、1-甲基-4-苯基-1,2,3,6-四氢吡啶（MPTP）和1-甲基-4-苯基吡啶离子（MPP+）进行定量分析。

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