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1

Retracted: PRP from Personal Blood Relieves Joint Fluid-Inducing Synovial Injury through NF-B Pathway and Mitochondrial Apoptosis in Human Synovial Fibroblast Cells.撤回：来自个人血液的富血小板血浆通过NF-κB途径和线粒体凋亡减轻人滑膜成纤维细胞中关节液诱导的滑膜损伤。

Biomed Res Int. 2023 Aug 2;2023:9875202. doi: 10.1155/2023/9875202. eCollection 2023.

2

PRP from Personal Blood Relieves Joint Fluid-Inducing Synovial Injury through NF-B Pathway and Mitochondrial Apoptosis in Human Synovial Fibroblast Cells.富含血小板血浆（PRP）通过核因子-κB（NF-κB）通路和人滑膜成纤维细胞线粒体凋亡缓解关节液诱导性滑膜损伤。

Biomed Res Int. 2022 Oct 10;2022:4496949. doi: 10.1155/2022/4496949. eCollection 2022.

3

Retracted: MSN@IL-4 Sustainingly Mediates Macrophagocyte M2 Polarization and Relieves Osteoblast Damage via NF-B Pathway-Associated Apoptosis.撤回：MSN@IL-4 通过 NF-κB 通路相关凋亡持续介导巨噬细胞 M2 极化并减轻成骨细胞损伤。

Biomed Res Int. 2024 Jan 9;2024:9784781. doi: 10.1155/2024/9784781. eCollection 2024.

4

Therapeutic Effect of Penta-acetyl Geniposide on Adjuvant-Induced Arthritis in Rats: Involvement of Inducing Synovial Apoptosis and Inhibiting NF-κB Signal Pathway.五乙酰栀子苷对大鼠佐剂性关节炎的治疗作用：诱导滑膜细胞凋亡和抑制 NF-κB 信号通路的参与。

Inflammation. 2018 Dec;41(6):2184-2195. doi: 10.1007/s10753-018-0861-0.

5

Eugenol suppresses the proliferation and invasion of TNF-α-induced fibroblast-like synoviocytes via regulating NF-κB and COX-2.丁香酚通过调节核因子κB和环氧化酶-2抑制肿瘤坏死因子-α诱导的成纤维样滑膜细胞的增殖和侵袭。

Biochem Biophys Res Commun. 2022 Jul 5;612:63-69. doi: 10.1016/j.bbrc.2022.04.074. Epub 2022 Apr 26.

6

Retracted: Study on the Mechanism of Platelet-Released Clusterins Inducing Restenosis after Carotid Endarterectomy by Activating TLR3/NF-b p65 Signaling Pathway.撤稿：血小板释放的簇集素通过激活TLR3/NF-κB p65信号通路诱导颈动脉内膜切除术后再狭窄的机制研究。

J Healthc Eng. 2023 Feb 2;2023:9784573. doi: 10.1155/2023/9784573. eCollection 2023.

7

Leishmanial lipid suppresses tumor necrosis factor alpha, interleukin-1beta, and nitric oxide production by adherent synovial fluid mononuclear cells in rheumatoid arthritis patients and induces apoptosis through the mitochondrial-mediated pathway.利什曼原虫脂质可抑制类风湿性关节炎患者滑膜液单核细胞产生肿瘤坏死因子α、白细胞介素-1β和一氧化氮，并通过线粒体介导的途径诱导细胞凋亡。

Arthritis Rheum. 2008 Mar;58(3):696-706. doi: 10.1002/art.23295.

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Vasoactive intestinal peptide ameliorates synovial cell functions of collagen-induced arthritis rats by down-regulating NF-kappaB activity.血管活性肠肽通过下调核因子κB活性改善胶原诱导性关节炎大鼠的滑膜细胞功能。

Immunol Invest. 2005;34(2):153-69.

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Inhibition of miR-16 enhances the sensitivity of fibroblast-like synovial cells to methotrexate by restraining MDR1/P-gp expression NF-κB pathway.抑制miR-16通过抑制MDR1/P-gp表达的NF-κB途径增强成纤维样滑膜细胞对甲氨蝶呤的敏感性。

RSC Adv. 2019 Aug 27;9(46):26619-26627. doi: 10.1039/c9ra04991f. eCollection 2019 Aug 23.

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Effect of IL-1β on apoptosis of synovial cells in rheumatoid arthritis rats via the NF-κB pathway.白细胞介素-1β 通过 NF-κB 通路对类风湿关节炎大鼠滑膜细胞凋亡的影响。

Eur Rev Med Pharmacol Sci. 2019 Dec;23(23):10211-10217. doi: 10.26355/eurrev_201912_19656.

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撤回：来自个人血液的富血小板血浆通过NF-κB途径和线粒体凋亡减轻人滑膜成纤维细胞中关节液诱导的滑膜损伤。

Retracted: PRP from Personal Blood Relieves Joint Fluid-Inducing Synovial Injury through NF-B Pathway and Mitochondrial Apoptosis in Human Synovial Fibroblast Cells.

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