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肱骨髁上骨突:正中神经高度受压的罕见原因。

Supracondylar Apophysis of the Humerus: Rare Cause of High Compression of the Median Nerve.

作者信息

Vital Luísa, Vidinha Vitor, Neves Nuno, Negrão Pedro

机构信息

Médicos Ortopedistas, Departamento de Ortopedia e Traumatologia, Centro Hospitalar Universitário de São João, Porto, Portugal.

出版信息

Rev Bras Ortop (Sao Paulo). 2020 Oct 29;58(4):e659-e661. doi: 10.1055/s-0040-1718509. eCollection 2023 Aug.

DOI:10.1055/s-0040-1718509
PMID:37663176
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10468235/
Abstract

Supracondylar apophysis (SA) is a bony prominence that originates from the anteromedial aspect of the distal humerus with a lower projection and which, although usually asymptomatic, due to the relationship with adjacent structures can cause symptoms. We describe the case of a 42-year-old woman with pain complaints radiating from her elbow to her hand, with 6 months of evolution. On objective examination, the patient had a sensory deficit in the median nerve territory and decreased grip strength. Radiographs of the distal humerus were performed, in which a bone spike was visible, and magnetic resonance imaging showed thickening of the median nerve epineurium. Electromyography showed severe axonal demyelination of the median nerve proximal to the elbow. A median nerve compression caused by a SA was diagnosed. The patient underwent surgery and, 1 year after the operation, she had a complete clinical recovery. Supracondylar apophysis is a rare, but possible and treatable cause of high median nerve compression.

摘要

肱骨髁上骨突(SA)是一种骨隆起,起源于肱骨远端的前内侧,突出较低,尽管通常无症状,但由于与相邻结构的关系可能会引起症状。我们描述了一名42岁女性的病例,她主诉肘部至手部有放射痛,病程6个月。体格检查时,患者在正中神经分布区域存在感觉障碍,握力下降。对肱骨远端进行了X线检查,可见骨棘,磁共振成像显示正中神经神经外膜增厚。肌电图显示肘部近端正中神经严重轴索性脱髓鞘。诊断为SA导致的正中神经受压。患者接受了手术,术后1年临床完全恢复。肱骨髁上骨突是正中神经高位受压的一种罕见但可治的病因。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/5f53e57f1e24/10-1055-s-0040-1718509-i2000131pt-3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/48c43987f52c/10-1055-s-0040-1718509-i2000131en-1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/067f7df9ebc9/10-1055-s-0040-1718509-i2000131en-2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/6e37a63abad8/10-1055-s-0040-1718509-i2000131en-3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/43e9e6941df4/10-1055-s-0040-1718509-i2000131pt-1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/f2dca1c15779/10-1055-s-0040-1718509-i2000131pt-2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/5f53e57f1e24/10-1055-s-0040-1718509-i2000131pt-3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/48c43987f52c/10-1055-s-0040-1718509-i2000131en-1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/067f7df9ebc9/10-1055-s-0040-1718509-i2000131en-2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/6e37a63abad8/10-1055-s-0040-1718509-i2000131en-3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/43e9e6941df4/10-1055-s-0040-1718509-i2000131pt-1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/f2dca1c15779/10-1055-s-0040-1718509-i2000131pt-2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4ebe/10468235/5f53e57f1e24/10-1055-s-0040-1718509-i2000131pt-3.jpg

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J Pain Res. 2018 Apr 16;11:803-807. doi: 10.2147/JPR.S160861. eCollection 2018.
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