Department of Anatomy, Jagiellonian University Medical College, International Evidence-Based Anatomy Working Group, Kraków, Poland.
Artromedical Orthopedic Clinic, Antracytowa 1, 97-400, Bełchatów, Poland.
Int Orthop. 2024 Feb;48(2):401-408. doi: 10.1007/s00264-023-05961-0. Epub 2023 Sep 5.
External snapping hip syndrome (ESHS) was historically attributed to isolated iliotibial band (ITB) contracture. However, the gluteus maximus complex (GMC) may also be involved. This study aimed to intraoperatively identify the ESHS origin and assess the outcomes of endoscopic treatment based on the identified aetiological type.
From 2008-2014, 30 consecutive patients (34 hips) with symptomatic ESHS cases refractory to conservative treatment underwent endoscopic stepwise "fan-like" release, gradually addressing all known reasons of ESHS: from the isolated ITB, through the fascial part of the GMC until a partial release of gluteus maximus femoral attachment occurred. Snapping was assessed intra-operatively after each surgical step and prospectively recorded. Functional outcomes were assessed via the MAHORN Hip Outcome Tool (MHOT-14).
Twenty seven patients (31 hips) were available to follow-up at 24-56 months. In all cases, complete snapping resolution was achieved intra-operatively: in seven cases (22.6%) after isolated ITB release, in 22 cases (70.9%), after release of ITB + fascial part of the GMC, and in two cases (6.5%) after ITB + fascial GMC release + partial release of GM femoral insertion. At follow-up, there were no snapping recurrences and MHOT-14 score significantly increased from a pre-operative average of 46 to 93(p<0.001).
Intraoperative identification and gradual addressing of all known causes of ESHS allows for maximum preservation of surrounding tissue during surgery while precisely targeting the directly involved structures. Endoscopic stepwise "fan-like" release of the ITB and GMC is an effective, tailor-made treatment option for ESHS regardless of the snapping origin in the patients with possibility to manually reproduce the snapping.
外弹响髋综合征(ESHS)历史上归因于阔筋膜张肌(ITB)挛缩。然而,臀大肌复合体(GMC)也可能参与其中。本研究旨在术中确定 ESHS 起源,并根据确定的病因类型评估内镜治疗的结果。
2008 年至 2014 年,30 例(34 髋)症状性 ESHS 患者经保守治疗无效,接受了内镜分步“扇形”松解术,逐步解决所有已知的 ESHS 原因:从单独的 ITB,到 GMC 的筋膜部分,直到发生臀大肌股骨附着部分松解。术中评估并前瞻性记录每次手术步骤后的弹响情况。通过 MAHORN 髋关节结局工具(MHOT-14)评估功能结局。
27 例(31 髋)患者可随访 24-56 个月。在所有病例中,术中均完全消除了弹响:7 例(22.6%)行 ITB 松解后,22 例(70.9%)行 ITB+GMC 筋膜部分松解后,2 例(6.5%)行 ITB+GMC 筋膜松解+臀大肌股骨附着部分松解后。随访时,无弹响复发,MHOT-14 评分从术前平均 46 分显著增加到 93 分(p<0.001)。
术中识别和逐步解决所有已知的 ESHS 原因,可以在手术过程中最大限度地保留周围组织,同时精确针对直接受累结构。内镜分步“扇形”松解 ITB 和 GMC 是一种有效、个体化的治疗 ESHS 的方法,无论患者弹响的起源如何,都可以手动再现弹响。