Caldwell W E
Genet Soc Gen Psychol Monogr. 1986 Aug;112(3):325-41.
An apnea hypothesis is proposed termed Conditioned Hyperventilation. An organism (animal, infant, or adult) can become conditioned to anticipate traumatic terminal breathing. The resulting hyperventilation produces excessive oxygen, which triggers apnea, allowing an increase in carbon dioxide. This apnea serves as an additional unconditioned stimulus (UCS) causing the hyperventilation (apnea) to increase. Organic blockage can also serve as the UCS. Apnea may be reduced or prevented by monitoring and extinguishing hyperventilation through the use of behavioral modification techniques, or biofeedback. Infant lung collapse during the first six months is suggested as one UCS in infants.
提出了一种名为条件性过度通气的呼吸暂停假说。生物体（动物、婴儿或成人）可能会形成条件反射，预期到创伤性的临终呼吸。由此产生的过度通气会产生过多氧气，从而引发呼吸暂停，使二氧化碳增加。这种呼吸暂停充当了额外的非条件刺激（UCS），导致过度通气（呼吸暂停）加剧。器质性阻塞也可作为非条件刺激。通过使用行为矫正技术或生物反馈来监测和消除过度通气，呼吸暂停可能会减轻或预防。婴儿出生后头六个月的肺萎陷被认为是婴儿的一种非条件刺激。
