Baldissera F, Cantarella G, Marini G, Ottaviani F
Laryngoscope. 1986 Dec;96(12):1399-404. doi: 10.1288/00005537-198612000-00017.
The aim of this work was to reestablish the respiratory function of the paralyzed larynx through reinnervation of the posterior cricoarytenoid (PCA) muscle by phrenic motoneurons. In nine adult cats the adductor branch of the recurrent laryngeal nerve (RLN) of one side was cut and ligated, while the abductor branch was left intact. The whole RLN was then transected lower in the neck and its distal stump anastomosed to the upper branch of the phrenic nerve. Periodical laryngoscopies under ketamine anesthesia assessed that the inspiratory abduction of the paralyzed vocal cord recovered within 45 days to 60 days in all cats. Abduction was caused by reinnervation of the PCA muscle from phrenic motoneurons, as demonstrated by electrophysiological and anatomical (retrograde transport of horseradish peroxidase) testings.
这项工作的目的是通过膈运动神经元对环杓后肌（PCA）进行再支配，从而恢复麻痹喉部的呼吸功能。在9只成年猫中，一侧喉返神经（RLN）的内收肌支被切断并结扎，而外展肌支保持完整。然后在颈部较低位置横断整个RLN，并将其远端残端与膈神经的上支进行吻合。在氯胺酮麻醉下定期进行喉镜检查评估，所有猫的麻痹声带吸气性外展在45天至60天内恢复。电生理和解剖学（辣根过氧化物酶逆行运输）测试表明，外展是由膈运动神经元对PCA肌肉的再支配引起的。
