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本文引用的文献

1
Lack of S1PR2 in Macrophage Ameliorates Sepsis-associated Lung Injury through Inducing IL-33-mediated Type 2 Immunity.缺乏 S1PR2 的巨噬细胞通过诱导 IL-33 介导体液免疫来改善脓毒症相关性肺损伤。
Am J Respir Cell Mol Biol. 2024 Mar;70(3):215-225. doi: 10.1165/rcmb.2023-0075OC.
2
IL-33-mediated Eosinophilia Protects against Acute Lung Injury.白细胞介素-33介导的嗜酸性粒细胞增多可预防急性肺损伤。
Am J Respir Cell Mol Biol. 2021 May;64(5):569-578. doi: 10.1165/rcmb.2020-0166OC.
3
Heterogeneity in the initiation, development and function of type 2 immunity.2 型免疫的启动、发展和功能的异质性。
Nat Rev Immunol. 2020 Oct;20(10):603-614. doi: 10.1038/s41577-020-0301-x. Epub 2020 May 4.
4
Protection against Staphylococcus aureus bacteremia-induced mortality depends on ILC2s and eosinophils.抵抗金黄色葡萄球菌菌血症诱导的死亡率依赖于 ILC2s 和嗜酸性粒细胞。
JCI Insight. 2019 Mar 21;4(6). doi: 10.1172/jci.insight.124168.
5
Blockage of sphingosine-1-phosphate receptor 2 attenuates allergic asthma in mice.阻断鞘氨醇-1-磷酸受体 2 可减轻小鼠过敏性哮喘。
Br J Pharmacol. 2019 Apr;176(7):938-949. doi: 10.1111/bph.14597. Epub 2019 Mar 14.
6
Integrating molecular pathogenesis and clinical translation in sepsis-induced acute respiratory distress syndrome.脓毒症诱导的急性呼吸窘迫综合征中分子发病机制与临床转化的整合
JCI Insight. 2019 Jan 24;4(2):e124061. doi: 10.1172/jci.insight.124061.
7
Sphingosine-1-phosphate Receptor 2 Signaling Promotes Caspase-11-dependent Macrophage Pyroptosis and Worsens Escherichia coli Sepsis Outcome.鞘氨醇-1-磷酸受体 2 信号促进 caspase-11 依赖性巨噬细胞焦亡，并加重大肠埃希菌脓毒症的结局。
Anesthesiology. 2018 Aug;129(2):311-320. doi: 10.1097/ALN.0000000000002196.
8
Interleukin-33 (IL-33): A nuclear cytokine from the IL-1 family.白细胞介素-33（IL-33）：IL-1 家族的一种核细胞因子。
Immunol Rev. 2018 Jan;281(1):154-168. doi: 10.1111/imr.12619.
9
Immunity by equilibrium.平衡免疫。
Nat Rev Immunol. 2016 Aug;16(8):524-32. doi: 10.1038/nri.2016.75. Epub 2016 Jul 11.
10
Sphingosine 1-phosphate Receptor 2 Signaling Suppresses Macrophage Phagocytosis and Impairs Host Defense against Sepsis.鞘氨醇 1-磷酸受体 2 信号抑制巨噬细胞吞噬作用并损害宿主对败血症的防御。
Anesthesiology. 2015 Aug;123(2):409-22. doi: 10.1097/ALN.0000000000000725.

S1PR2: A Fulcrum in the Balance of Type 1 and Type 2 Responses during Sepsis-induced Acute Lung Injury.

作者信息

Verhoef Philip A

机构信息

John A. Burns School of Medicine University of Hawaii Honolulu, Hawaii and Hawaii Permanente Medical Group Honolulu, Hawaii.

出版信息

Am J Respir Cell Mol Biol. 2024 Mar;70(3):157-158. doi: 10.1165/rcmb.2023-0433ED.

DOI:10.1165/rcmb.2023-0433ED
PMID:38226863
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10914765/
Abstract
摘要