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撤回:组蛋白去乙酰化酶9通过Toll样受体4调控代表M1巨噬细胞极化和炎症反应的表观遗传促进作用。

Retracted: Histone Deacetylase9 Represents the Epigenetic Promotion of M1 Macrophage Polarization and Inflammatory Response via TLR4 Regulation.

作者信息

International BioMed Research

出版信息

Biomed Res Int. 2024 Jan 9;2024:9820109. doi: 10.1155/2024/9820109. eCollection 2024.

DOI:10.1155/2024/9820109
PMID:38229980
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10791340/
Abstract

[This retracts the article DOI: 10.1155/2022/7408136.].

摘要

[本文撤回文章DOI: 10.1155/2022/7408136。]

相似文献

1
Retracted: Histone Deacetylase9 Represents the Epigenetic Promotion of M1 Macrophage Polarization and Inflammatory Response via TLR4 Regulation.撤回:组蛋白去乙酰化酶9通过Toll样受体4调控代表M1巨噬细胞极化和炎症反应的表观遗传促进作用。
Biomed Res Int. 2024 Jan 9;2024:9820109. doi: 10.1155/2024/9820109. eCollection 2024.
2
Histone Deacetylase9 Represents the Epigenetic Promotion of M1 Macrophage Polarization and Inflammatory Response via TLR4 Regulation.组蛋白去乙酰化酶 9 通过 TLR4 调节介导 M1 巨噬细胞极化和炎症反应的表观遗传促进作用。
Biomed Res Int. 2022 Jul 30;2022:7408136. doi: 10.1155/2022/7408136. eCollection 2022.
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Hsa_circRNA_103124 upregulation in Crohn's disease promoted macrophage M1 polarization to maintain an inflammatory microenvironment via activation of the AKT2 and TLR4/NF-κB pathways.Hsa_circRNA_103124 在克罗恩病中的上调通过激活 AKT2 和 TLR4/NF-κB 通路促进巨噬细胞 M1 极化,以维持炎症微环境。
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Retracted: Qingchang Wenzhong Decoction Alleviates DSS-Induced Inflammatory Bowel Disease by Inhibiting M1 Macrophage Polarization and .撤回:清肠温中汤通过抑制M1巨噬细胞极化减轻右旋糖酐硫酸钠诱导的炎症性肠病及…… (原文此处不完整)
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Retracted: Heparin-Binding Protein Aggravates Acute Lung Injury in Septic Rats by Promoting Macrophage M1 Polarization and NF-B Signaling Pathway Activation.撤回:肝素结合蛋白通过促进巨噬细胞M1极化和NF-κB信号通路激活加重脓毒症大鼠急性肺损伤
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本文引用的文献

1
Histone Deacetylase9 Represents the Epigenetic Promotion of M1 Macrophage Polarization and Inflammatory Response via TLR4 Regulation.组蛋白去乙酰化酶 9 通过 TLR4 调节介导 M1 巨噬细胞极化和炎症反应的表观遗传促进作用。
Biomed Res Int. 2022 Jul 30;2022:7408136. doi: 10.1155/2022/7408136. eCollection 2022.