The inhibition of several components of terminal complement pathway results in C3 binding to PNH red blood cells.
作者信息
Sica Michela, Notaro Rosario
机构信息
Laboratory of Cancer Genetics and Gene Transfer, Core Research Laboratory, Istituto per lo Studio, la Prevenzione e la Rete Oncologica (ISPRO), Florence, Italy.
出版信息
Am J Hematol. 2024 Mar;99(3):505-507. doi: 10.1002/ajh.27215. Epub 2024 Jan 24.
PMID:38264841
Abstract
C3 binding on PNH red blood cells after in vitro complement activation in normal and terminal complement depleted (∆5, ∆6, ∆9) sera: C3 binding, and the subsequent extravascular hemolysis, happens anytime there is a block of a component of the terminal complement pathway.
摘要
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