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病例报告:局灶性房性心动过速患者出现广泛分裂的P波。

Case report: Widely split P' waves in a patient with focal atrial tachycardia.

作者信息

Jiang Hao, Ruan Zhongbao, Ren Yin, Ding Xiangwei

机构信息

Department of Cardiology, The Affiliated Taizhou People's Hospital of Nanjing Medical University, Taizhou School of Clinical Medicine, Nanjing Medical University, Taizhou, China.

出版信息

Front Cardiovasc Med. 2024 Jan 11;10:1303200. doi: 10.3389/fcvm.2023.1303200. eCollection 2023.

Abstract

BACKGROUND

Widely split P waves in sinus rhythm have been reported previously. However, widely split P' waves in focal atrial tachycardia (AT) on a surface electrocardiogram (ECG) have rarely been reported. The electrophysiological mechanism is relatively difficult to clarify, requiring a electrophysiological study.

CASE SUMMARY

A 67-year-old patient, who had undergone two radiofrequency ablations for atrial fibrillation, presented with recurrent palpitation. During the palpitation episode, the 12-lead ECG showed AT with a 3:1 atrioventricular conduction rate. P' waves were markedly prolonged in duration and widely split in morphology. An electrophysiological study showed that the tachycardia arose from the left atrial appendage (LAA) and was conducted through two distinct pathways. The impulse of one pathway was transmitted solely via the superior part of the atrium, including the Bachmann bundle. The second pathway was conducted via the coronary sinus and transmitted the impulse from the LAA to the ventricle. After the site showed that the earliest activation was ablated, repeated intravenous infusion of isoprenaline and programmed atrial stimulation did not induce tachycardia.

CONCLUSION

Widely split P' waves in AT indicate intra- and interatrial conduction blocks, which can be easily overlooked due to the presence of low-voltage areas. Therefore, an electrophysiological study is crucial for identifying the origin of the tachycardia and elucidating the mechanistic details.

摘要

背景

先前已有窦性心律时P波广泛分裂的报道。然而,体表心电图(ECG)上局灶性房性心动过速(AT)时P′波广泛分裂的情况鲜有报道。其电生理机制相对难以阐明,需要进行电生理研究。

病例摘要

一名67岁患者,曾因心房颤动接受过两次射频消融治疗,现出现反复发作的心悸。心悸发作时,12导联心电图显示为房室传导比例3:1的房性心动过速。P′波时限明显延长,形态上广泛分裂。电生理研究显示,心动过速起源于左心耳(LAA),并通过两条不同的途径传导。一条途径的冲动仅通过心房上部,包括Bachmann束传导。第二条途径通过冠状窦传导,将冲动从左心耳传至心室。在最早激动部位消融后,重复静脉注射异丙肾上腺素和程控心房刺激均未诱发心动过速。

结论

房性心动过速时P′波广泛分裂提示心房内和心房间传导阻滞,由于存在低电压区域,这些情况容易被忽视。因此,电生理研究对于确定心动过速的起源和阐明机制细节至关重要。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b08f/10808459/e389a1f125cd/fcvm-10-1303200-g001.jpg

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