Seshadri M S, Cornish C J, Mason R S, Posen S
Clin Endocrinol (Oxf). 1985 Dec;23(6):689-97. doi: 10.1111/j.1365-2265.1985.tb01130.x.
Extracts from the tumours of three patients with oncogenic osteomalacia stimulated renal adenylate cyclase in a parathyroid hormone (PTH) responsive system. The stimulation was suppressed by a specific PTH antagonist. These observations suggest that osteomalacia-producing tumours exert some of their effects through PTH-like material acting on PTH receptors.
三名致癌性骨软化症患者肿瘤提取物，在甲状旁腺激素（PTH）反应系统中刺激了肾腺苷酸环化酶。这种刺激被一种特异性PTH拮抗剂所抑制。这些观察结果表明，产生骨软化症的肿瘤通过作用于PTH受体的类PTH物质发挥其部分作用。
