Correction: Reduction of NgR in perforant path decreases amyloid-β peptide production and ameliorates synaptic and cognitive deficits in APP/PS1 mice.
作者信息
Jiang Rong, Wu Xue-Fei, Wang Bin, Guan Rong-Xiao, Lv Lang-Man, Li Ai-Ping, Lei Lei, Ma Ye, Li Na, Li Qi-Fa, Ma Quan-Hong, Zhao Jie, Li Shao
机构信息
Liaoning Provincial Key Laboratory of Cerebral Diseases, Department of Physiology, College of Basic Medical Sciences, Dalian Medical University, Dalian, China.
National-Local Joint Engineering Research Center for Drug Research and Development (R&D) of Neurodegenerative Diseases, Dalian Medical University, Dalian, China.
出版信息
Alzheimers Res Ther. 2024 Feb 27;16(1):47. doi: 10.1186/s13195-024-01389-9.
Abstract
摘要
相似文献
1
Correction: Reduction of NgR in perforant path decreases amyloid-β peptide production and ameliorates synaptic and cognitive deficits in APP/PS1 mice.更正：降低穿孔通路中的NgR可减少淀粉样β肽的产生，并改善APP/PS1小鼠的突触和认知缺陷。
Alzheimers Res Ther. 2024 Feb 27;16(1):47. doi: 10.1186/s13195-024-01389-9.
2
Reduction of NgR in perforant path decreases amyloid-β peptide production and ameliorates synaptic and cognitive deficits in APP/PS1 mice.NgR 表达减少可降低淀粉样β肽的产生，并改善 APP/PS1 小鼠的突触和认知功能障碍。
Alzheimers Res Ther. 2020 Apr 24;12(1):47. doi: 10.1186/s13195-020-00616-3.
3
Reduction of NgR in perforant path protects neuronal morphology and function in APP/PS1 transgenic mice.NgR 表达减少可保护 APP/PS1 转基因小鼠的颗粒细胞层神经元形态和功能。
Aging (Albany NY). 2023 Mar 23;15(6):2158-2169. doi: 10.18632/aging.204605.
4
Huatuo Zaizao pill ameliorates cognitive impairment of APP/PS1 transgenic mice by improving synaptic plasticity and reducing Aβ deposition.华佗再造丸通过改善突触可塑性和减少 Aβ 沉积改善 APP/PS1 转基因小鼠的认知障碍。
BMC Complement Altern Med. 2018 May 29;18(1):167. doi: 10.1186/s12906-018-2237-2.
5
Muscone Ameliorates Synaptic Dysfunction and Cognitive Deficits in APP/PS1 Mice.麝香酮改善 APP/PS1 小鼠的突触功能障碍和认知缺陷。
J Alzheimers Dis. 2020;76(2):491-504. doi: 10.3233/JAD-200188.
6
The blockage of the Nogo/NgR signal pathway in microglia alleviates the formation of Aβ plaques and tau phosphorylation in APP/PS1 transgenic mice.小胶质细胞中Nogo/NgR信号通路的阻断可减轻APP/PS1转基因小鼠中Aβ斑块的形成和tau蛋白磷酸化。
J Neuroinflammation. 2016 Mar 3;13(1):56. doi: 10.1186/s12974-016-0522-x.
7
The Amyloid Precursor Protein Regulates Synaptic Transmission at Medial Perforant Path Synapses.淀粉样前体蛋白调节内侧穿通纤维突触的突触传递。
J Neurosci. 2023 Jul 19;43(29):5290-5304. doi: 10.1523/JNEUROSCI.1824-22.2023. Epub 2023 Jun 27.
8
Danggui Buxue decoction ameliorates mitochondrial biogenesis and cognitive deficits through upregulating histone H4 lysine 12 acetylation in APP/PS1 mice.当归补血汤通过上调 APP/PS1 小鼠组蛋白 H4 赖氨酸 12 乙酰化来改善线粒体生物发生和认知缺陷。
J Ethnopharmacol. 2023 Sep 15;313:116554. doi: 10.1016/j.jep.2023.116554. Epub 2023 May 1.
9
Overexpression of human apolipoprotein A-I preserves cognitive function and attenuates neuroinflammation and cerebral amyloid angiopathy in a mouse model of Alzheimer disease.载脂蛋白 A-I 过表达可保护认知功能，并减轻阿尔茨海默病小鼠模型的神经炎症和脑淀粉样血管病。
J Biol Chem. 2010 Nov 19;285(47):36958-68. doi: 10.1074/jbc.M110.127829. Epub 2010 Sep 16.
10
Lamotrigine attenuates deficits in synaptic plasticity and accumulation of amyloid plaques in APP/PS1 transgenic mice.拉莫三嗪可减轻APP/PS1转基因小鼠的突触可塑性缺陷和淀粉样斑块积累。
Neurobiol Aging. 2014 Dec;35(12):2713-2725. doi: 10.1016/j.neurobiolaging.2014.06.009. Epub 2014 Jun 16.
本文引用的文献
1
Reduction of NgR in perforant path decreases amyloid-β peptide production and ameliorates synaptic and cognitive deficits in APP/PS1 mice.NgR 表达减少可降低淀粉样β肽的产生，并改善 APP/PS1 小鼠的突触和认知功能障碍。
Alzheimers Res Ther. 2020 Apr 24;12(1):47. doi: 10.1186/s13195-020-00616-3.
Zotero 插件